Interleukin-1 Induces the Release of Lubricating Phospholipids from Human Osteoarthritic Fibroblast-like Synoviocytes.
ATP Binding Cassette Transporter 1
/ genetics
Benzoates
/ pharmacology
Benzylamines
/ pharmacology
Cells, Cultured
Cytochrome P450 Family 7
/ genetics
Gene Expression Regulation
/ drug effects
Humans
Interleukin-1beta
/ pharmacology
Liver X Receptors
/ genetics
Osteoarthritis
/ genetics
Phospholipids
/ metabolism
Steroid Hydroxylases
/ genetics
Synovial Fluid
/ cytology
Synoviocytes
/ cytology
ABCA1
CH25H
CYP7B1
FLS
LXR
cholesterol hydroxylase
interleukin
osteoarthritis
phospholipids
synovial fibroblasts
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
22 Feb 2022
22 Feb 2022
Historique:
received:
21
01
2022
revised:
09
02
2022
accepted:
19
02
2022
entrez:
10
3
2022
pubmed:
11
3
2022
medline:
26
3
2022
Statut:
epublish
Résumé
(1) Background: Synovial fluid (SF) from knee joints with osteoarthritis (OA) has increased levels of phospholipids (PL). We have reported earlier that TGF-ß and IGF-1 stimulate fibroblast-like synoviocytes (FLS) to synthesize increased amounts of PLs. The current study examined whether IL-1ß induces the release of PLs in FLS and the underlying mechanism. (2) Methods: Cultured human OA FLS were treated with IL-1ß alone and with pathway inhibitors or with synthetic liver X receptor (LXR) agonists. Cholesterol hydroxylases, ABC transporters, apolipoproteins (APO), LXR, sterol regulatory binding proteins (SREBPs), and 3-hydroxy-3-methylglutaryl-coenzyme A reductase (HMGCR) were analyzed by RT-PCR, Western blot, and ELISA. The release of radiolabeled PLs from FLS was determined, and statistical analysis was performed using R (N = 5-9). (3) Results: Like synthetic LXR agonists, IL-1ß induced a 1.4-fold greater release of PLs from FLS. Simultaneously, IL-1ß upregulated the level of the PL transporter ABCA1 and of cholesterol hydroxylases CH25H and CYP7B1. IL-1ß and T0901317 stimulated the expression of SREBP1c, whereas only T0901317 enhanced SREBP2, HMGCR, APOE, LXRα, and ABCG1 additionally. (4) Conclusions: IL-1ß partially controls PL levels in OA-SF by affecting the release of PLs from FLS. Our data show that IL-1ß upregulates cholesterol hydroxylases and thus the formation of oxysterols, which, as natural agonists of LXR, increase the level of active ABCA1, in turn enhancing the release of PLs.
Identifiants
pubmed: 35269552
pii: ijms23052409
doi: 10.3390/ijms23052409
pmc: PMC8910712
pii:
doi:
Substances chimiques
ABCA1 protein, human
0
ATP Binding Cassette Transporter 1
0
Benzoates
0
Benzylamines
0
GW 3965
0
Interleukin-1beta
0
Liver X Receptors
0
Phospholipids
0
Steroid Hydroxylases
EC 1.14.-
Cytochrome P450 Family 7
EC 1.14.14.23
CYP7B1 protein, human
EC 1.14.14.29
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : DRB Foundation
ID : 39/2017
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