Deterioration after Liver Transplantation and Transthyretin Stabilizer Administration in a Patient with ATTRv Amyloidosis with a Leu58Arg (p.Leu78Arg) TTR Variant.


Journal

Internal medicine (Tokyo, Japan)
ISSN: 1349-7235
Titre abrégé: Intern Med
Pays: Japan
ID NLM: 9204241

Informations de publication

Date de publication:
01 Aug 2022
Historique:
pubmed: 15 3 2022
medline: 3 8 2022
entrez: 14 3 2022
Statut: ppublish

Résumé

We herein report a 44-year-old Japanese man with hereditary transthyretin amyloidosis (ATTRv amyloidosis) harboring the variant Leu58Arg (p.Leu78Arg) in TTR in whom we conducted an observational study with liver transplantation (LT) and transthyretin (TTR) stabilizers (tafamidis and diflunisal) for 9 years. This patient showed gradual deterioration of sensory, motor, and autonomic neuropathy symptoms after LT. Furthermore, cardiac amyloidosis gradually developed. Although the present case showed deterioration of the symptoms after disease-modifying treatments, LT might be suitable in patients with the same variant if they are young and in good condition due to a long survival after LT.

Identifiants

pubmed: 35283385
doi: 10.2169/internalmedicine.8945-21
pmc: PMC9424092
doi:

Substances chimiques

Prealbumin 0

Types de publication

Case Reports Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

2347-2351

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Auteurs

Sadao Hikishima (S)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Kenji Sakai (K)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Akio Akagi (A)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Hiroki Yamaguchi (H)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Shutaro Shibata (S)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Koji Hayashi (K)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Hiroto Nakano (H)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Mizuki Kanemoto (M)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Yuta Usui (Y)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Yu Taniguchi (Y)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Junji Komatsu (J)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Keiko Nakamura-Shindo (K)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Ichiro Nozaki (I)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Tsuyoshi Hamaguchi (T)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Kenjiro Ono (K)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Kazuo Iwasa (K)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

Masahito Yamada (M)

Department of Neurology and Neurobiology of Aging, Kanazawa University Graduate School of Medical Sciences, Japan.

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