Development and External Validation of a Model to Predict Complex Treatment After Radiofrequency Ablation for Barrett's Esophagus With Early Neoplasia.


Journal

Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association
ISSN: 1542-7714
Titre abrégé: Clin Gastroenterol Hepatol
Pays: United States
ID NLM: 101160775

Informations de publication

Date de publication:
11 2022
Historique:
received: 08 12 2021
revised: 25 01 2022
accepted: 16 02 2022
pubmed: 17 3 2022
medline: 2 11 2022
entrez: 16 3 2022
Statut: ppublish

Résumé

Endoscopic eradication therapy for Barrett's esophagus (BE)-related neoplasia is safe and leads to complete eradication in the majority of patients. However, a subgroup will experience a more complex treatment course with a risk for failure or disease progression. Early identification of these patients may improve patient counseling and treatment outcomes. We aimed to develop a prognostic model for a complex treatment course. We collected data from a nationwide registry that captures outcomes for all patients undergoing endoscopic eradication therapy for early BE neoplasia. A complex treatment course was defined as neoplastic progression, treatment failure, or the need for endoscopic resection during the radiofrequency ablation treatment phase. We developed a prognostic model using logistic regression. We externally validated our model in an independent registry. A total of 1386 patients were included, of whom 78 (6%) had a complex treatment course. Our model identified patients with a BE length of 9 cm or longer with a visible lesion containing high-grade dysplasia/cancer, and patients with less than 50% squamous conversion after radiofrequency ablation were identified as high risk for a complex treatment. This applied to 8% of the study population and included 93% of all treatment failures and 76% of all patients with advanced neoplastic progression. The model appeared robust in multiple sensitivity analyses and performed well in external validation (area under the curve, 0.84). We developed a prognostic model that identified patients with a BE length of 9 cm or longer and high-grade dysplasia/esophageal adenocarcinoma and those with poor squamous regeneration as high risk for a complex treatment course. The good performance in external validation suggests that it may be used in clinical management (Netherlands Trial Register: NL7039).

Sections du résumé

BACKGROUND & AIMS
Endoscopic eradication therapy for Barrett's esophagus (BE)-related neoplasia is safe and leads to complete eradication in the majority of patients. However, a subgroup will experience a more complex treatment course with a risk for failure or disease progression. Early identification of these patients may improve patient counseling and treatment outcomes. We aimed to develop a prognostic model for a complex treatment course.
METHODS
We collected data from a nationwide registry that captures outcomes for all patients undergoing endoscopic eradication therapy for early BE neoplasia. A complex treatment course was defined as neoplastic progression, treatment failure, or the need for endoscopic resection during the radiofrequency ablation treatment phase. We developed a prognostic model using logistic regression. We externally validated our model in an independent registry.
RESULTS
A total of 1386 patients were included, of whom 78 (6%) had a complex treatment course. Our model identified patients with a BE length of 9 cm or longer with a visible lesion containing high-grade dysplasia/cancer, and patients with less than 50% squamous conversion after radiofrequency ablation were identified as high risk for a complex treatment. This applied to 8% of the study population and included 93% of all treatment failures and 76% of all patients with advanced neoplastic progression. The model appeared robust in multiple sensitivity analyses and performed well in external validation (area under the curve, 0.84).
CONCLUSIONS
We developed a prognostic model that identified patients with a BE length of 9 cm or longer and high-grade dysplasia/esophageal adenocarcinoma and those with poor squamous regeneration as high risk for a complex treatment course. The good performance in external validation suggests that it may be used in clinical management (Netherlands Trial Register: NL7039).

Identifiants

pubmed: 35292379
pii: S1542-3565(22)00246-4
doi: 10.1016/j.cgh.2022.02.057
pii:
doi:

Banques de données

NTR
['NL7039']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2495-2504.e5

Informations de copyright

Copyright © 2022 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Sanne N van Munster (SN)

Department of Gastroenterology and Hepatology, Amsterdam Gastroenterology Endocrinology and Metabolism, Amsterdam University Medical Centers, Amsterdam, The Netherlands; Department of Gastroenterology and Hepatology, Sint Antonius Hospital, Nieuwegein, The Netherlands.

Esther Nieuwenhuis (E)

Department of Gastroenterology and Hepatology, Sint Antonius Hospital, Nieuwegein, The Netherlands.

Raf Bisschops (R)

Department of Gastroenterology and Hepatology, University Hospitals Leuven, Koninklijke Universiteit Leuven, Belgium.

Hilde Willekens (H)

Department of Gastroenterology and Hepatology, University Hospitals Leuven, Koninklijke Universiteit Leuven, Belgium.

Bas L A M Weusten (BLAM)

Department of Gastroenterology and Hepatology, Sint Antonius Hospital, Nieuwegein, The Netherlands; Department of Gastroenterology and Hepatology, University Medical Center Utrecht, Utrecht University, Utrecht, The Netherlands.

Lorenza Alvarez Herrero (LA)

Department of Gastroenterology and Hepatology, Sint Antonius Hospital, Nieuwegein, The Netherlands.

Auke Bogte (A)

Department of Gastroenterology and Hepatology, University Medical Center Utrecht, Utrecht University, Utrecht, The Netherlands.

Alaa Alkhalaf (A)

Department of Gastroenterology and Hepatology, Isala Clinics, Zwolle, The Netherlands.

Ed B E Schenk (EBE)

Department of Gastroenterology and Hepatology, Isala Clinics, Zwolle, The Netherlands.

Erik J Schoon (EJ)

Department of Gastroenterology and Hepatology, Catharina Hospital, Eindhoven, The Netherlands; GROW School for Oncology and Developmental Biology, Faculty of Health, Medicine and Life Sciences, Maastricht University, Maastricht, The Netherlands.

Wouter Curvers (W)

Department of Gastroenterology and Hepatology, Catharina Hospital, Eindhoven, The Netherlands.

Arjun D Koch (AD)

Department of Gastroenterology and Hepatology, Erasmus MC Cancer Institute, University Medical Center Rotterdam, Rotterdam, The Netherlands.

Pieter Jan F de Jonge (PJF)

Department of Gastroenterology and Hepatology, Erasmus MC Cancer Institute, University Medical Center Rotterdam, Rotterdam, The Netherlands.

Tjon J Tang (TJ)

Department of Gastroenterology and Hepatology, IJsselland Hospital, Cappelle aan den Ijssel, The Netherlands.

Wouter B Nagengast (WB)

Department of Gastroenterology and Hepatology, University Medical Center Groningen, Groningen University, Groningen, The Netherlands.

Jessie Westerhof (J)

Department of Gastroenterology and Hepatology, University Medical Center Groningen, Groningen University, Groningen, The Netherlands.

Martin H M G Houben (MHMG)

Department of Gastroenterology and Hepatology, Haga Teaching Hospital, Den Haag, The Netherlands.

Jacques J G H M Bergman (JJGHM)

Department of Gastroenterology and Hepatology, Amsterdam Gastroenterology Endocrinology and Metabolism, Amsterdam University Medical Centers, Amsterdam, The Netherlands. Electronic address: j.bergman@amsterdamumc.nl.

Roos E Pouw (RE)

Department of Gastroenterology and Hepatology, Amsterdam Gastroenterology Endocrinology and Metabolism, Amsterdam University Medical Centers, Amsterdam, The Netherlands.

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