Childhood Threat Is Associated With Lower Resting-State Connectivity Within a Central Visceral Network.
affective disorders
amygdala
bed nucleus of stria terminalis
childhood trauma
extended amygdala
hypothalamus
subgenual anterior cingulate cortex
Journal
Frontiers in psychology
ISSN: 1664-1078
Titre abrégé: Front Psychol
Pays: Switzerland
ID NLM: 101550902
Informations de publication
Date de publication:
2022
2022
Historique:
received:
29
10
2021
accepted:
09
02
2022
entrez:
21
3
2022
pubmed:
22
3
2022
medline:
22
3
2022
Statut:
epublish
Résumé
Childhood adversity is associated with altered or dysregulated stress reactivity; these altered patterns of physiological functioning persist into adulthood. Evidence from both preclinical animal models and human neuroimaging studies indicates that early life experience differentially influences stressor-evoked activity within central visceral neural circuits proximally involved in the control of stress responses, including the subgenual anterior cingulate cortex (sgACC), paraventricular nucleus of the hypothalamus (PVN), bed nucleus of the stria terminalis (BNST) and amygdala. However, the relationship between childhood adversity and the resting-state connectivity of this central visceral network remains unclear. To this end, we examined relationships between childhood threat and childhood socioeconomic deprivation, the resting-state connectivity between our regions of interest (ROIs), and affective symptom severity and diagnoses. We recruited a transdiagnostic sample of young adult males and females (
Identifiants
pubmed: 35310241
doi: 10.3389/fpsyg.2022.805049
pmc: PMC8927539
doi:
Types de publication
Journal Article
Langues
eng
Pagination
805049Informations de copyright
Copyright © 2022 Banihashemi, Peng, Rangarajan, Karim, Wallace, Sibbach, Singh, Stinley, Germain and Aizenstein.
Déclaration de conflit d'intérêts
MW is a statistical consultant for Noctem, unrelated to this work. AG serves as CEO and holds equity in Rehat, LLC and has also served as a consultant for Jazz Pharmaceuticals, Inc., unrelated to this work. The remaining authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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