International Tumor Budding Consensus Conference criteria determine the prognosis of oesophageal adenocarcinoma with poor response to neoadjuvant treatment.

Adenocarcinoma Neoadjuvant therapy Oesophagus Prognosis Tumour budding

Journal

Pathology, research and practice
ISSN: 1618-0631
Titre abrégé: Pathol Res Pract
Pays: Germany
ID NLM: 7806109

Informations de publication

Date de publication:
Apr 2022
Historique:
received: 21 01 2022
accepted: 13 03 2022
pubmed: 23 3 2022
medline: 6 4 2022
entrez: 22 3 2022
Statut: ppublish

Résumé

Neoadjuvant therapy regimens followed by surgery represent the current standard treatment of locally advanced oesophageal adenocarcinomas. Tumour regression determines prognosis, but more than half of patients do have more than 10% residual tumour after neoadjuvant therapy. In these cases, classical histopathological parameters for the determination of prognosis are of limited value. Therefore, we investigated whether tumour budding could be an additional prognostic factor for tumours with poor response to neoadjuvant therapy. Tumour budding was assessed according to a standardized consensus quantification method as proposed by the International Tumor Budding Consensus Conference (ITBCC) in H&E-stained whole tissue slides of 278 formalin-fixed paraffin-embedded (FFPE) resected oesophageal adenocarcinomas with a poor response (> 10% vital residual tumour) to neoadjuvant therapy. We could demonstrate a strong positive correlation (p < 0.05) between the budding group, ypN stage and UICC tumour stage. Further, high numbers of tumour buds were a significant and independent negative prognostic marker for OS in all studied patients (HR = 1.039 (95% CI 1.012-1.066), p = 0.004). ITBCC budding groups were an independent prognostic parameter. Tumour budding assessed in accordance with the ITBCC criteria may aid in the prognostic stratification of locally advanced oesophageal adenocarcinoma with poor response to neoadjuvant treatment.

Sections du résumé

BACKGROUND BACKGROUND
Neoadjuvant therapy regimens followed by surgery represent the current standard treatment of locally advanced oesophageal adenocarcinomas. Tumour regression determines prognosis, but more than half of patients do have more than 10% residual tumour after neoadjuvant therapy. In these cases, classical histopathological parameters for the determination of prognosis are of limited value. Therefore, we investigated whether tumour budding could be an additional prognostic factor for tumours with poor response to neoadjuvant therapy.
METHODS METHODS
Tumour budding was assessed according to a standardized consensus quantification method as proposed by the International Tumor Budding Consensus Conference (ITBCC) in H&E-stained whole tissue slides of 278 formalin-fixed paraffin-embedded (FFPE) resected oesophageal adenocarcinomas with a poor response (> 10% vital residual tumour) to neoadjuvant therapy.
RESULTS RESULTS
We could demonstrate a strong positive correlation (p < 0.05) between the budding group, ypN stage and UICC tumour stage. Further, high numbers of tumour buds were a significant and independent negative prognostic marker for OS in all studied patients (HR = 1.039 (95% CI 1.012-1.066), p = 0.004). ITBCC budding groups were an independent prognostic parameter.
CONCLUSIONS CONCLUSIONS
Tumour budding assessed in accordance with the ITBCC criteria may aid in the prognostic stratification of locally advanced oesophageal adenocarcinoma with poor response to neoadjuvant treatment.

Identifiants

pubmed: 35316631
pii: S0344-0338(22)00087-5
doi: 10.1016/j.prp.2022.153844
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

153844

Informations de copyright

Copyright © 2022 Elsevier GmbH. All rights reserved.

Auteurs

Philipp Lohneis (P)

University of Cologne Faculty of Medicine and University Hospital Cologne, Institute of Pathology, Kerpener Strasse 62, D-50924 Cologne, Germany. Electronic address: lohneis@hp-hl.de.

Julia Rohmann (J)

University of Cologne Faculty of Medicine and University Hospital Cologne, Institute of Pathology, Kerpener Strasse 62, D-50924 Cologne, Germany.

Florian Gebauer (F)

University of Cologne Faculty of Medicine and University Hospital Cologne, Department of General, Visceral, Cancer and Transplantation Surgery, Kerpener Strasse 62, D-50924 Cologne, Germany.

Lena Hieggelke (L)

University of Cologne Faculty of Medicine and University Hospital Cologne, Institute of Pathology, Kerpener Strasse 62, D-50924 Cologne, Germany.

Christiane Bruns (C)

University of Cologne Faculty of Medicine and University Hospital Cologne, Department of General, Visceral, Cancer and Transplantation Surgery, Kerpener Strasse 62, D-50924 Cologne, Germany.

Wolfgang Schröder (W)

University of Cologne Faculty of Medicine and University Hospital Cologne, Department of General, Visceral, Cancer and Transplantation Surgery, Kerpener Strasse 62, D-50924 Cologne, Germany.

Reinhard Büttner (R)

University of Cologne Faculty of Medicine and University Hospital Cologne, Institute of Pathology, Kerpener Strasse 62, D-50924 Cologne, Germany.

Heike Löser (H)

University of Cologne Faculty of Medicine and University Hospital Cologne, Institute of Pathology, Kerpener Strasse 62, D-50924 Cologne, Germany.

Alexander Quaas (A)

University of Cologne Faculty of Medicine and University Hospital Cologne, Institute of Pathology, Kerpener Strasse 62, D-50924 Cologne, Germany.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH