The Fra-1/AP-1 Oncoprotein: From the "Undruggable" Transcription Factor to Therapeutic Targeting.

AP-1 complex FOSL1 Fos-Related-Antigen-1 therapeutic targeting transcription factor

Journal

Cancers
ISSN: 2072-6694
Titre abrégé: Cancers (Basel)
Pays: Switzerland
ID NLM: 101526829

Informations de publication

Date de publication:
14 Mar 2022
Historique:
received: 04 02 2022
revised: 04 03 2022
accepted: 10 03 2022
entrez: 25 3 2022
pubmed: 26 3 2022
medline: 26 3 2022
Statut: epublish

Résumé

The genetic and epigenetic changes affecting transcription factors, coactivators, and chromatin modifiers are key determinants of the hallmarks of cancer. The acquired dependence on oncogenic transcriptional regulators, representing a major determinant of cancer cell vulnerability, points to transcription factors as ideal therapeutic targets. However, given the unavailability of catalytic activities or binding pockets for small-molecule inhibitors, transcription factors are generally regarded as undruggable proteins. Among components of the AP-1 complex, the FOS-family transcription factor Fra-1, encoded by

Identifiants

pubmed: 35326630
pii: cancers14061480
doi: 10.3390/cancers14061480
pmc: PMC8946526
pii:
doi:

Types de publication

Journal Article Review

Langues

eng

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Auteurs

Laura Casalino (L)

Institute of Genetics and Biophysics "Adriano Buzzati Traverso", Consiglio Nazionale dele Ricerche (CNR), 80131 Naples, Italy.

Francesco Talotta (F)

ReiThera Srl, 00128 Rome, Italy.

Amelia Cimmino (A)

Institute of Genetics and Biophysics "Adriano Buzzati Traverso", Consiglio Nazionale dele Ricerche (CNR), 80131 Naples, Italy.

Pasquale Verde (P)

Institute of Genetics and Biophysics "Adriano Buzzati Traverso", Consiglio Nazionale dele Ricerche (CNR), 80131 Naples, Italy.

Classifications MeSH