Blood M2-like Monocyte Polarization Is Associated with Calcific Plaque Phenotype in Stable Coronary Artery Disease: A Sub-Study of SMARTool Clinical Trial.

blood monocyte subsets coronary CT angiography coronary artery disease flow cytometry plaque calcium volume plasma cytokines

Journal

Biomedicines
ISSN: 2227-9059
Titre abrégé: Biomedicines
Pays: Switzerland
ID NLM: 101691304

Informations de publication

Date de publication:
28 Feb 2022
Historique:
received: 07 02 2022
revised: 23 02 2022
accepted: 24 02 2022
entrez: 25 3 2022
pubmed: 26 3 2022
medline: 26 3 2022
Statut: epublish

Résumé

Atherosclerosis is a chronic inflammatory disease. The balance between pro- and anti-inflammatory factors, acting on the arterial wall, promotes less or more coronary plaque macro-calcification, respectively. We investigated the association between monocyte phenotypic polarization and CTCA-assessed plaque dense-calcium volume (DCV) in patients with stable coronary artery disease (CAD). In 55 patients, individual DCV component was assessed by quantitative CTCA and normalized to total plaque volume. Flow cytometry expression of CD14, CD16, CD18, CD11b, HLA-DR, CD163, CCR2, CCR5, CX3CR1 and CXCR4 was quantified. Adhesion molecules and cytokines were measured by ELISA. DCV values were significantly associated, by multiple regression analysis, with the expression (RFI) of CCR5 ( The association between DCV and M2-like phenotypic polarization of circulating monocytes indicates that plaque macro-calcification in stable CAD may be partly modulated by an anti-inflammatory monocyte functional state, as evidenced by cell membrane receptor patterns.

Sections du résumé

BACKGROUND BACKGROUND
Atherosclerosis is a chronic inflammatory disease. The balance between pro- and anti-inflammatory factors, acting on the arterial wall, promotes less or more coronary plaque macro-calcification, respectively. We investigated the association between monocyte phenotypic polarization and CTCA-assessed plaque dense-calcium volume (DCV) in patients with stable coronary artery disease (CAD).
METHODS METHODS
In 55 patients, individual DCV component was assessed by quantitative CTCA and normalized to total plaque volume. Flow cytometry expression of CD14, CD16, CD18, CD11b, HLA-DR, CD163, CCR2, CCR5, CX3CR1 and CXCR4 was quantified. Adhesion molecules and cytokines were measured by ELISA.
RESULTS RESULTS
DCV values were significantly associated, by multiple regression analysis, with the expression (RFI) of CCR5 (
CONCLUSIONS CONCLUSIONS
The association between DCV and M2-like phenotypic polarization of circulating monocytes indicates that plaque macro-calcification in stable CAD may be partly modulated by an anti-inflammatory monocyte functional state, as evidenced by cell membrane receptor patterns.

Identifiants

pubmed: 35327367
pii: biomedicines10030565
doi: 10.3390/biomedicines10030565
pmc: PMC8945688
pii:
doi:

Types de publication

Journal Article

Langues

eng

Subventions

Organisme : European Commission
ID : 689068

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Auteurs

Silverio Sbrana (S)

CNR Institute of Clinical Physiology, 54100 Massa, Italy.

Antonella Cecchettini (A)

Department of Clinical and Experimental Medicine, University of Pisa, 56126 Pisa, Italy.
CNR Institute of Clinical Physiology, 56124 Pisa, Italy.

Luca Bastiani (L)

CNR Institute of Clinical Physiology, 54100 Massa, Italy.

Nicoletta Di Giorgi (N)

CNR Institute of Clinical Physiology, 56124 Pisa, Italy.

Annamaria Mazzone (A)

Fondazione Toscana Gabriele Monasterio, 54100 Massa, Italy.

Elisa Ceccherini (E)

CNR Institute of Clinical Physiology, 56124 Pisa, Italy.

Federico Vozzi (F)

CNR Institute of Clinical Physiology, 56124 Pisa, Italy.

Chiara Caselli (C)

CNR Institute of Clinical Physiology, 56124 Pisa, Italy.

Danilo Neglia (D)

Fondazione Toscana Gabriele Monasterio, 56124 Pisa, Italy.

Alberto Clemente (A)

Fondazione Toscana Gabriele Monasterio, 54100 Massa, Italy.

Arthur J H A Scholte (AJHA)

Department of Cardiology, Leiden University Medical Center, 2333 ZA Leiden, The Netherlands.

Oberdan Parodi (O)

CNR Institute of Clinical Physiology, 56124 Pisa, Italy.

Gualtiero Pelosi (G)

CNR Institute of Clinical Physiology, 56124 Pisa, Italy.

Silvia Rocchiccioli (S)

CNR Institute of Clinical Physiology, 56124 Pisa, Italy.

Classifications MeSH