From the Structural and (Dys)Function of ATP Synthase to Deficiency in Age-Related Diseases.

ATP synthase cell death mitochondria neurodegenerative diseases

Journal

Life (Basel, Switzerland)
ISSN: 2075-1729
Titre abrégé: Life (Basel)
Pays: Switzerland
ID NLM: 101580444

Informations de publication

Date de publication:
10 Mar 2022
Historique:
received: 15 02 2022
revised: 25 02 2022
accepted: 08 03 2022
entrez: 25 3 2022
pubmed: 26 3 2022
medline: 26 3 2022
Statut: epublish

Résumé

The ATP synthase is a mitochondrial inner membrane complex whose function is essential for cell bioenergy, being responsible for the conversion of ADP into ATP and playing a role in mitochondrial

Identifiants

pubmed: 35330152
pii: life12030401
doi: 10.3390/life12030401
pmc: PMC8949411
pii:
doi:

Types de publication

Journal Article Review

Langues

eng

Subventions

Organisme : Italian Minister of University and Research - Rita Levi Montalcini Program - Rientro cervelli
ID : RLM2017
Organisme : CARISBO Foundation
ID : 2021.0058
Organisme : CARISBO Foundation
ID : 2021.0173

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Auteurs

Caterina Garone (C)

Department of Medical and Surgical Sciences, Alma Mater Studiorum University of Bologna, 40137 Bologna, Italy.
Center for Applied Biomedical Research, Alma Mater Studiorum University of Bologna, 40137 Bologna, Italy.
UOC Neuropsichiatria dell'età Pediatrica, IRCCS Istituto delle Scienze Neurologiche di Bologna, 40137 Bologna, Italy.

Andrea Pietra (A)

Department of Medical and Surgical Sciences, Alma Mater Studiorum University of Bologna, 40137 Bologna, Italy.
UO Genetica Medica, IRCCS Azienda Ospedaliero-Universitaria di Bologna, 40137 Bologna, Italy.

Salvatore Nesci (S)

Department of Veterinary Medical Sciences, Alma Mater Studiorum University of Bologna, 40064 Ozzano Emilia, Italy.

Classifications MeSH