Human Polymorphonuclear Cells Support Zika Virus to Cross Endothelial Monolayer and Access Bloodstream.
HUVECs
Zika virus
cell junctions
endothelial monolayer
viral infection
Journal
Pathogens (Basel, Switzerland)
ISSN: 2076-0817
Titre abrégé: Pathogens
Pays: Switzerland
ID NLM: 101596317
Informations de publication
Date de publication:
05 Mar 2022
05 Mar 2022
Historique:
received:
14
01
2022
revised:
27
02
2022
accepted:
03
03
2022
entrez:
26
3
2022
pubmed:
27
3
2022
medline:
27
3
2022
Statut:
epublish
Résumé
The rapid spread of new outbreaks of human infection caused by Zika virus (ZIKV) has raised many global concerns since 2016. Despite the increasing knowledge of this virus, data on the pathogenesis of ZIKV are still missing. In particular, it is still unknown how the virus crosses the endothelial monolayer and gets access to the bloodstream. In the present work, we used human umbilical vein endothelial cells (HUVECs) as a model to study ZIKV infection
Identifiants
pubmed: 35335645
pii: pathogens11030321
doi: 10.3390/pathogens11030321
pmc: PMC8955922
pii:
doi:
Types de publication
Journal Article
Langues
eng
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