Detailed videocapillaroscopic microvascular changes detectable in adult COVID-19 survivors.

COVID-19 Capillaries Endothelial cells Glucocorticoids Microcirculation Nailfold capillaroscopy Raynaud's phenomenon SARS-CoV-2

Journal

Microvascular research
ISSN: 1095-9319
Titre abrégé: Microvasc Res
Pays: United States
ID NLM: 0165035

Informations de publication

Date de publication:
07 2022
Historique:
received: 03 01 2022
revised: 08 03 2022
accepted: 14 03 2022
pubmed: 28 3 2022
medline: 7 6 2022
entrez: 27 3 2022
Statut: ppublish

Résumé

COVID-19 is a multisystem disease that causes endothelial dysfunction and organ damage. Aim of the study was to evaluate the microvascular status in COVID-19 survivors with past different disease severity, in comparison with age and sex-matched primary Raynaud's phenomenon (PRP) patients and control subjects (CNT), including possible effects of concomitant therapies. Sixty-one COVID-19 survivors (mean age 58 ± 13 years, mean days from disease onset 126 ± 53 and mean days from recovery 104 ± 53), thirty-one PRP patients (mean age 59 ± 15 years, mean disease duration 11 ± 10 years) and thirty CNT (mean age 58 ± 13 years) underwent nailfold videocapillaroscopy (NVC) examination. The following capillaroscopic parameters were searched and scored (0-3): dilated capillaries, giant capillaries, isolated microhemorrhages, capillary ramifications (angiogenesis) and capillary number, including absolute capillary number per linear millimeter at the nailfold bed. The mean nailfold capillary number per linear millimeter was significantly lower in COVID-19 survivors when compared with PRP patients and CNT (univariate and multivariate analysis p < 0.001). On the contrary, COVID-19 survivors showed significantly less isolated microhemorrhages than PRP patients and CNT (univariate and multivariate analysis, p = 0.005 and p = 0.012, respectively). No statistically significant difference was observed between COVID-19 survivors and control groups concerning the frequency of dilated capillaries and capillary ramifications. COVID-19 selective therapies showed a promising trend on preserving capillary loss and deserving further investigations. SARS-CoV-2 seems to mainly induce a significant loss of capillaries in COVID-19 survivors at detailed NVC analysis in comparison to controls. The presence of a significant reduced score for isolated microhaemorrhages in COVID-19 survivors deserves further analysis.

Identifiants

pubmed: 35339493
pii: S0026-2862(22)00051-6
doi: 10.1016/j.mvr.2022.104361
pmc: PMC8942583
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

104361

Informations de copyright

Copyright © 2022 Elsevier Inc. All rights reserved.

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Auteurs

Alberto Sulli (A)

Laboratory of Experimental Rheumatology and Academic Division of Clinical Rheumatology, Department of Internal Medicine and Specialties, University of Genova, IRCCS San Martino Polyclinic Hospital, Genova, Italy. Electronic address: albertosulli@unige.it.

Emanuele Gotelli (E)

Laboratory of Experimental Rheumatology and Academic Division of Clinical Rheumatology, Department of Internal Medicine and Specialties, University of Genova, IRCCS San Martino Polyclinic Hospital, Genova, Italy.

Pietro Francesco Bica (PF)

Laboratory of Experimental Rheumatology and Academic Division of Clinical Rheumatology, Department of Internal Medicine and Specialties, University of Genova, IRCCS San Martino Polyclinic Hospital, Genova, Italy.

Irene Schiavetti (I)

Department of Health Sciences, University of Genova, Genova, Italy.

Carmen Pizzorni (C)

Laboratory of Experimental Rheumatology and Academic Division of Clinical Rheumatology, Department of Internal Medicine and Specialties, University of Genova, IRCCS San Martino Polyclinic Hospital, Genova, Italy. Electronic address: carmen.pizzorni@unige.it.

Teresita Aloè (T)

Interventional Pneumology Unit, IRCCS San Martino Polyclinic Hospital, Genova, Italy.

Marco Grosso (M)

Interventional Pneumology Unit, IRCCS San Martino Polyclinic Hospital, Genova, Italy.

Emanuela Barisione (E)

Interventional Pneumology Unit, IRCCS San Martino Polyclinic Hospital, Genova, Italy.

Sabrina Paolino (S)

Laboratory of Experimental Rheumatology and Academic Division of Clinical Rheumatology, Department of Internal Medicine and Specialties, University of Genova, IRCCS San Martino Polyclinic Hospital, Genova, Italy. Electronic address: sabrina.paolino@unige.it.

Vanessa Smith (V)

Department of Rheumatology, Ghent University Hospital, Ghent, Belgium; Department of Internal Medicine, Ghent University Hospital, Ghent, Belgium; Unit for Molecular Immunology and Inflammation, Vlaams Instituut voor Biotechnologie (VIB), Inflammation Research Center (IRC), Ghent, Belgium. Electronic address: vanessa.smith@ugent.be.

Maurizio Cutolo (M)

Laboratory of Experimental Rheumatology and Academic Division of Clinical Rheumatology, Department of Internal Medicine and Specialties, University of Genova, IRCCS San Martino Polyclinic Hospital, Genova, Italy. Electronic address: mcutolo@unige.it.

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