Protective effect of edaravone on cisplatin-induced injury in rat ovary.


Journal

Archives of gynecology and obstetrics
ISSN: 1432-0711
Titre abrégé: Arch Gynecol Obstet
Pays: Germany
ID NLM: 8710213

Informations de publication

Date de publication:
11 2022
Historique:
received: 31 10 2021
accepted: 16 03 2022
pubmed: 1 4 2022
medline: 1 10 2022
entrez: 31 3 2022
Statut: ppublish

Résumé

This study was aimed to evaluate the protective effect of edaravone on cisplatin-induced ovarian injury. A total 40 female Wistar-Albino rats were utilized to form four groups: Group 1 (control group) (n = 10), no procedure was performed. Group 2 (cisplatin group) (n = 10), single-dose 7.5 mg/kg cisplatin was administered and no procedure was performed. Group 3 (edaravone group) (n = 10), single-dose 1 mg/kg edaravone was administered and no procedure was performed. Group 4 (cisplatin + edaravone group) (n = 10), single-dose 7.5 mg/kg cisplatin and 1 mg/kg edaravone were administered. Seventy-two hours later, ovaries were surgically extirpated in all groups. Malondialdehyde (MDA) levels and nitric oxide (NO) levels were studied in blood samples. In ovarian tissue samples, DNA damage and apoptosis were assessed using TUNEL method. Ovarian tissue damage was evaluated by immunohistochemical staining with caspase 3 and caspase 8. According to the findings obtained from the study, edaravone showed protective properties on ovarian damage due to cisplatin. MDA and NO levels were significantly higher in cisplatin group than other groups. Histopathological ovarian tissue damage in the cisplatin group was significantly higher than other groups. Similarly, DNA damage and apoptosis were higher in cisplatin group and this difference was found to be statistically significant. The immunohistochemical staining which was done using caspase 3 and caspase 8 was revealed that immunoreactive cells were statistically higher in cisplatin group than cisplatin + edaravone group. Edaravone seems to be effective in prevention of ovarian damage and short-term treatment.

Identifiants

pubmed: 35357583
doi: 10.1007/s00404-022-06538-9
pii: 10.1007/s00404-022-06538-9
doi:

Substances chimiques

Nitric Oxide 31C4KY9ESH
Malondialdehyde 4Y8F71G49Q
Caspase 3 EC 3.4.22.-
Caspase 8 EC 3.4.22.-
Cisplatin Q20Q21Q62J
Edaravone S798V6YJRP
Antipyrine T3CHA1B51H

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1673-1678

Informations de copyright

© 2022. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.

Références

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Auteurs

Ozlem Kara (O)

Department of Histology and Embryology, Kirsehir Ahi Evran University Medical Faculty, Bagbasi Mah, Şehit Necdet Yagiz Cad. No: 143/E, Merkez, Kirsehir, Turkey. ozlemozturk34@hotmail.com.

Emin Kaymak (E)

Department of Histology and Embryology, Yozgat Bozok University Medical Faculty, Yozgat, Turkey.

Birkan Yakan (B)

Department of Histology and Embryology, Erciyes University Institute of Health Sciences, Kayseri, Turkey.

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