Barrier-Forming Potential of Epithelial Cells from the Exstrophic Bladder.


Journal

The American journal of pathology
ISSN: 1525-2191
Titre abrégé: Am J Pathol
Pays: United States
ID NLM: 0370502

Informations de publication

Date de publication:
06 2022
Historique:
received: 23 12 2021
revised: 04 02 2022
accepted: 03 03 2022
pubmed: 1 4 2022
medline: 14 6 2022
entrez: 31 3 2022
Statut: ppublish

Résumé

Bladder exstrophy (BEX) is a rare developmental abnormality resulting in an open, exposed bladder plate. Although normal bladder urothelium is a mitotically quiescent barrier epithelium, histologic studies of BEX epithelia report squamous and proliferative changes that can persist beyond surgical closure. The current study examined whether patient-derived BEX epithelial cells in vitro were capable of generating a barrier-forming epithelium under permissive conditions. Epithelial cells isolated from 11 BEX samples, classified histologically as transitional (n = 6) or squamous (n = 5), were propagated in vitro. In conditions conducive to differentiated tight barrier formation by normal human urothelial cell cultures, 8 of 11 BEX lines developed transepithelial electrical resistances of more than 1000 Ω.cm

Identifiants

pubmed: 35358476
pii: S0002-9440(22)00109-2
doi: 10.1016/j.ajpath.2022.03.009
pmc: PMC9194657
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

943-955

Subventions

Organisme : Medical Research Council
ID : MR/N028414/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/L013347/1
Pays : United Kingdom
Organisme : Department of Health
Pays : United Kingdom

Informations de copyright

Copyright © 2022 American Society for Investigative Pathology. Published by Elsevier Inc. All rights reserved.

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Auteurs

Jennifer Hinley (J)

Jack Birch Unit for Molecular Carcinogenesis, Department of Biology and York Biomedical Research Institute, University of York, York, United Kingdom.

Rosalind Duke (R)

Jack Birch Unit for Molecular Carcinogenesis, Department of Biology and York Biomedical Research Institute, University of York, York, United Kingdom.

Jessica Jinks (J)

Jack Birch Unit for Molecular Carcinogenesis, Department of Biology and York Biomedical Research Institute, University of York, York, United Kingdom.

Jens Stahlschmidt (J)

Jack Birch Unit for Molecular Carcinogenesis, Department of Biology and York Biomedical Research Institute, University of York, York, United Kingdom; Department of Histopathology, St James's University Hospital, Leeds, United Kingdom.

David Keene (D)

Department of Paediatric Urology, Royal Manchester Children's Hospital, Manchester, United Kingdom.

Raimondo M Cervellione (RM)

Department of Paediatric Urology, Royal Manchester Children's Hospital, Manchester, United Kingdom.

Imran Mushtaq (I)

Department of Paediatric Urology, Great Ormond Street Hospital for Children National Health Service Trust, London, United Kingdom.

Paolo De Coppi (P)

Department of Paediatric Urology, Great Ormond Street Hospital for Children National Health Service Trust, London, United Kingdom; National Institute for Health and Care Research Biomedical Research Centre, University College London Great Ormond Street Institute of Child Health, London, United Kingdom. Electronic address: p.decoppi@ucl.ac.uk.

Massimo Garriboli (M)

National Institute for Health and Care Research Biomedical Research Centre, University College London Great Ormond Street Institute of Child Health, London, United Kingdom; Paediatric Urology, Evelina London Children's Hospital, London, United Kingdom.

Jennifer Southgate (J)

Jack Birch Unit for Molecular Carcinogenesis, Department of Biology and York Biomedical Research Institute, University of York, York, United Kingdom. Electronic address: j.southgate@york.ac.uk.

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Classifications MeSH