NRAS
Kaposiform lymphangiomatosis
Lymphatic anomaly
RASopathies
Vascular Anomaly
Vascular Malformation
Journal
Angiogenesis
ISSN: 1573-7209
Titre abrégé: Angiogenesis
Pays: Germany
ID NLM: 9814575
Informations de publication
Date de publication:
08 2022
08 2022
Historique:
received:
18
10
2021
accepted:
22
03
2022
pubmed:
9
4
2022
medline:
7
7
2022
entrez:
8
4
2022
Statut:
ppublish
Résumé
Somatic mutations in NRAS drive the pathogenesis of melanoma and other cancers but their role in vascular anomalies and specifically human endothelial cells is unclear. The goals of this study were to determine whether the somatic-activating NRAS
Identifiants
pubmed: 35391614
doi: 10.1007/s10456-022-09836-7
pii: 10.1007/s10456-022-09836-7
pmc: PMC9631803
mid: NIHMS1838751
doi:
Substances chimiques
Membrane Proteins
0
GTP Phosphohydrolases
EC 3.6.1.-
NRAS protein, human
EC 3.6.1.-
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Research Support, N.I.H., Extramural
Langues
eng
Sous-ensembles de citation
IM
Pagination
331-342Subventions
Organisme : NHLBI NIH HHS
ID : R01 HL117952
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL156866
Pays : United States
Organisme : NIH HHS
ID : R01 HL156866
Pays : United States
Informations de copyright
© 2022. The Author(s), under exclusive licence to Springer Nature B.V.
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