The new generation tyrosine kinase inhibitor improves the survival of chronic myeloid leukemia patients after allogeneic stem cell transplantation.


Journal

Hematological oncology
ISSN: 1099-1069
Titre abrégé: Hematol Oncol
Pays: England
ID NLM: 8307268

Informations de publication

Date de publication:
Aug 2022
Historique:
revised: 08 02 2022
received: 22 11 2021
accepted: 31 03 2022
pubmed: 9 4 2022
medline: 10 8 2022
entrez: 8 4 2022
Statut: ppublish

Résumé

The introduction of tyrosine kinase inhibitors (TKIs) in chronic myeloid leukemia (CML) treatment has dramatically improved the prognosis of CML patients and reduced the number of patients receiving allogeneic stem cell transplantation (allo-SCT). However, the impact of the newest-generation TKIs on the overall survival (OS) after allo-SCT has not been well described. To investigate the beneficial effects of TKIs on the prognosis after allo-SCT, we conducted a retrospective observational study using the Transplant Registry Unified Management Program database in Japan. We analyzed 1188 patients (male/female: 738/450; median age: 44 years; range: 16-75) who underwent their first allo-SCT between January 2001 and December 2018. We divided the patients into two groups according to the TKI treatment used before allo-SCT: group 1 was treated with the first generation TKI imatinib; group 2 was treated with the second generation TKIs nilotinib, dasatinib, or bosutinib and/or the third generation TKI ponatinib. We compared the post allo-SCT OS between the two groups. The 3-year OS rates (95%CI) of groups 1 and 2 were 59.3% (54.8%-63.5%) and 65.8% (61.6%-69.6%), respectively (p = 0.017). Multivariate analysis confirmed that group 2 had superior OS after allo-SCT compared to group 1 (p = 0.002). Other factors associated with superior prognosis were age ≤65, performance status (PS) 0/1, a 6/6 HLA-matched donor and chronic-phase (CP) disease status at allo-SCT. A subgroup analysis showed poor prognoses for patients who could not obtain a molecular response before allo-SCT and patients with positive T315I mutation in the BCR/ABL gene. In group 2, early allo-SCT was correlated with superior OS in patients with a blast-crisis disease status at allo-SCT (p = 0.001). The cumulative incidence of non-relapse mortality rate significantly decreased in group 2 (p = 0.0005). The post allo-SCT OS was improved both by pre- and post-management of allo-SCT and by the introduction of newer TKIs.

Identifiants

pubmed: 35394658
doi: 10.1002/hon.3000
doi:

Substances chimiques

Protein Kinase Inhibitors 0
Dasatinib RBZ1571X5H

Types de publication

Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

442-456

Subventions

Organisme : Japan Agency for Medical Research and Development
ID : 18ek0510023h0002

Informations de copyright

© 2022 John Wiley & Sons Ltd.

Références

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Auteurs

Yutaka Shimazu (Y)

Department of Hematology, Kyoto University Hospital, Kyoto, Japan.

Makoto Murata (M)

Department of Hematology and Oncology, Nagoya University Graduate School of Medicine, Nagoya, Aichi, Japan.

Takeshi Kondo (T)

Department of Hematology, Aiiku Hospital, Sapporo, Hokkaido, Japan.

Yosuke Minami (Y)

Department of Hematology, National Cancer Center Hospital East, Kashiwa, Chiba, Japan.

Takayoshi Tachibana (T)

Department of Hematology, Kanagawa Cancer Center, Yokohama, Kanagawa, Japan.

Noriko Doki (N)

Hematology Division, Tokyo Metropolitan Cancer and Infectious Diseases Center, Komagome Hospital, Tokyo, Japan.

Naoyuki Uchida (N)

Department of Hematology, Federation of National Public Service Personnel Mutual Aid Associations TORANOMON, Tokyo, Japan.

Yukiyasu Ozawa (Y)

Department of Hematology, Japanese Red Cross Aichi Medical Center Nagoya Daiichi Hospital, Nagoya, Aichi, Japan.

Shingo Yano (S)

Department of Internal Medicine, Division of Clinical Oncology and Hematology, the Jikei University School of Medicine, Tokyo, Japan.

Takahiro Fukuda (T)

Department of Hematopoietic Stem Cell Transplantation, National Cancer Center Hospital, Tokyo, Japan.

Jun Kato (J)

Department of Medicine, Division of Hematology, Keio University School of Medicine, Tokyo, Japan.

Takahide Ara (T)

Department of Hematology, Hokkaido University Hospital, Sapporo, Hokkaido, Japan.

Testuya Eto (T)

Department of Hematology, Hamanomachi Hospital, Fukuoka, Japan.

Jun Ishikawa (J)

Department of Hematology, Osaka International Cancer Institute, Osaka, Japan.

Hirohisa Nakamae (H)

Department of Hematology, Osaka City University Hospital, Osaka, Japan.

Junji Tanaka (J)

Department of Hematology, Tokyo Women's Medical University, Tokyo, Japan.

Tatsuo Ichinohe (T)

Department of Hematology and Oncology, Research Institute for Radiation Biology and Medicine, Hiroshima University, Hiroshima, Japan.

Yoshiko Atsuta (Y)

Japanese Data Center for Hematopoietic Cell Transplantation, Nagoya, Aichi, Japan.
Department of Registry Science for Transplant and Cellular Therapy, Aichi Medical University School of Medicine, Nagoya, Aichi, Japan.

Tokiko Nagamura-Inoue (T)

Department of Cell Processing and Transfusion, Department of Hematology/Oncology, Institute of Medical Science, the University of Tokyo, Tokyo, Japan.

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