Postoperative atrial fibrillation and atrial epicardial fat: Is there a link?

AF, Atrial Fibrillation AV, Aortic Valve BMI, Body Mass Index BSA, Body Surface Area CABG, Coronary Artery Bypass Graft CCTA, Coronary Computed Tomographic Angiography CM, Contrast Management CT, Computed Tomography Cardiac surgery Computed tomography scan EAT, Epicardial Adipose Tissue EAT-V, EAT-Volume Epicardial adipose tissue HU, Hounsfield Units LA, Left Atrial LAVI, LA Volume Index LVEF, Left Ventricular Ejection Fraction MYO, Myocardial MYO-V, MYO-Volume POAF, Post-Operative AF Postoperative atrial fibrillation ROI, Regions Of Interest SR, Sinus Rhythm kV, Kilovoltage ml, Millilitre

Journal

International journal of cardiology. Heart & vasculature
ISSN: 2352-9067
Titre abrégé: Int J Cardiol Heart Vasc
Pays: Ireland
ID NLM: 101649525

Informations de publication

Date de publication:
Apr 2022
Historique:
received: 29 12 2021
revised: 04 02 2022
accepted: 14 02 2022
entrez: 11 4 2022
pubmed: 12 4 2022
medline: 12 4 2022
Statut: epublish

Résumé

Atrial Epicardial Adipose Tissue (EAT) is presumably involved in the pathogenesis of atrial fibrillation (AF). The transient nature of postoperative AF (POAF) suggests that surgery-induced triggers provoke an unmasking of a pre-existent AF substrate. The aim is to investigate the association between the volume of EAT and the occurrence of POAF. We hypothesise that the likelihood of developing POAF is higher in patients with high compared to low left atrial (LA) EAT volumes. Quantification of LA EAT based on the Hounsfield Units using custom made software was performed on pre-operative coronary computed tomography angiography scans of patients who underwent cardiac surgery between 2009 and 2019. Patients with mitral valve disease were excluded. A total of 83 patients were included in this study (CABG = 34, aortic valve = 33, aorta ascendens n = 7, combination n = 9), of which 43 patients developed POAF. The EAT percentage in the LA wall nor indexed EAT volumes differed between patients with POAF and with sinus rhythm (all P > 0.05). In multivariable analysis, age and LA volume index (LAVI) were the only independent predictors for early POAF (OR: 1.076 and 1.056, respectively). As expected, advanced age and LAVI were independent predictors of POAF. However, the amount of local EAT was not associated with the occurrence of AF after cardiac surgery. This suggests that the role of EAT in POAF is rather limited, or that the association of EAT in the early phase of POAF is obscured by the dominance of surgical-induced triggers.

Sections du résumé

Background UNASSIGNED
Atrial Epicardial Adipose Tissue (EAT) is presumably involved in the pathogenesis of atrial fibrillation (AF). The transient nature of postoperative AF (POAF) suggests that surgery-induced triggers provoke an unmasking of a pre-existent AF substrate. The aim is to investigate the association between the volume of EAT and the occurrence of POAF. We hypothesise that the likelihood of developing POAF is higher in patients with high compared to low left atrial (LA) EAT volumes.
Methods UNASSIGNED
Quantification of LA EAT based on the Hounsfield Units using custom made software was performed on pre-operative coronary computed tomography angiography scans of patients who underwent cardiac surgery between 2009 and 2019. Patients with mitral valve disease were excluded.
Results UNASSIGNED
A total of 83 patients were included in this study (CABG = 34, aortic valve = 33, aorta ascendens n = 7, combination n = 9), of which 43 patients developed POAF. The EAT percentage in the LA wall nor indexed EAT volumes differed between patients with POAF and with sinus rhythm (all P > 0.05). In multivariable analysis, age and LA volume index (LAVI) were the only independent predictors for early POAF (OR: 1.076 and 1.056, respectively).
Conclusions UNASSIGNED
As expected, advanced age and LAVI were independent predictors of POAF. However, the amount of local EAT was not associated with the occurrence of AF after cardiac surgery. This suggests that the role of EAT in POAF is rather limited, or that the association of EAT in the early phase of POAF is obscured by the dominance of surgical-induced triggers.

Identifiants

pubmed: 35402690
doi: 10.1016/j.ijcha.2022.100976
pii: S2352-9067(22)00025-2
pmc: PMC8984634
doi:

Types de publication

Journal Article

Langues

eng

Pagination

100976

Informations de copyright

© 2022 The Authors.

Déclaration de conflit d'intérêts

The authors report no relationships that could be construed as a conflict of interest.

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Auteurs

Claudia A J van der Heijden (CAJ)

Department of Cardiothoracic Surgery, Maastricht University Medical Centre, Maastricht, the Netherlands.

Sander Verheule (S)

Department of Physiology, Maastricht University, Maastricht, the Netherlands.
Cardiovascular Research Institute Maastricht (CARIM), Maastricht, the Netherlands.

Jules R Olsthoorn (JR)

Department of Cardiothoracic Surgery, Catharina Hospital Eindhoven, Eindhoven, the Netherlands.

Casper Mihl (C)

Cardiovascular Research Institute Maastricht (CARIM), Maastricht, the Netherlands.
Department of Radiology and Nuclear Medicine, Maastricht University Medical Centre, Maastricht, the Netherlands.

Lexan Poulina (L)

Department of Cardiothoracic Surgery, Maastricht University Medical Centre, Maastricht, the Netherlands.

Sander M J van Kuijk (SMJ)

Department of Clinical Epidemiology and Medical Technology Assessment, Maastricht University Medical Centre, Maastricht, the Netherlands.

Samuel Heuts (S)

Department of Cardiothoracic Surgery, Maastricht University Medical Centre, Maastricht, the Netherlands.

Jos G Maessen (JG)

Department of Cardiothoracic Surgery, Maastricht University Medical Centre, Maastricht, the Netherlands.
Cardiovascular Research Institute Maastricht (CARIM), Maastricht, the Netherlands.

Elham Bidar (E)

Department of Cardiothoracic Surgery, Maastricht University Medical Centre, Maastricht, the Netherlands.
Cardiovascular Research Institute Maastricht (CARIM), Maastricht, the Netherlands.

Bart Maesen (B)

Department of Cardiothoracic Surgery, Maastricht University Medical Centre, Maastricht, the Netherlands.
Cardiovascular Research Institute Maastricht (CARIM), Maastricht, the Netherlands.

Classifications MeSH