Gliadin Peptide P31-43 Induces mTOR/NFkβ Activation and Reduces Autophagy: The Role of
Lactobacillus paracasei CBA L74 postbiotc
celiac disease (CD)
gliadin peptide P31-43
mTOR/NFkβ activation
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
26 Mar 2022
26 Mar 2022
Historique:
received:
10
02
2022
revised:
18
03
2022
accepted:
24
03
2022
entrez:
12
4
2022
pubmed:
13
4
2022
medline:
14
4
2022
Statut:
epublish
Résumé
Celiac disease (CD) is an autoimmune disease characterized by an altered immune response stimulated by gliadin peptides that are not digested and cause damage to the intestinal mucosa. The aim of this study was to investigate whether the postbiotic
Identifiants
pubmed: 35409015
pii: ijms23073655
doi: 10.3390/ijms23073655
pmc: PMC8999065
pii:
doi:
Substances chimiques
Peptide Fragments
0
Peptides
0
Gliadin
9007-90-3
MTOR protein, human
EC 2.7.1.1
TOR Serine-Threonine Kinases
EC 2.7.11.1
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Italian Association for Celiac Disease (AIC)
ID : Grant 005_FC_2016
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