Berberine Modulates Macrophage Activation by Inducing Glycolysis.


Journal

Journal of immunology (Baltimore, Md. : 1950)
ISSN: 1550-6606
Titre abrégé: J Immunol
Pays: United States
ID NLM: 2985117R

Informations de publication

Date de publication:
15 05 2022
Historique:
received: 19 07 2021
accepted: 28 02 2022
pubmed: 17 4 2022
medline: 20 5 2022
entrez: 16 4 2022
Statut: ppublish

Résumé

Classical activation of macrophage and monocyte differentiation induced by β-glucan is accompanied with metabolic change in glucose. However, the role of the metabolic rewiring in monocyte/macrophage activation remains elusive. In this study, we show that berberine induces aerobic glycolysis by blocking the tricarboxylic acid cycle and modulates cytokine responses in bone marrow-derived macrophages (BMDMs) from mice and human PBMC. 13-Methyberberine had activities on glucose metabolism and BMDM activation similar to those of berberine, whereas other tested derivatives lost both activities. Glucose transporter (GLUT)1 expression and total cellular hexokinase activity increased gradually in BMDMs in the presence of berberine. In the contrast, LPS upregulated GLUT1 and 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase 3 (PFKFB3) levels in 6 h. Extracellular glucose levels and replacing glucose with galactose in culture medium affected the cytokine secretion of BMDMs. Berberine alleviated enteritis of

Identifiants

pubmed: 35428692
pii: jimmunol.2100716
doi: 10.4049/jimmunol.2100716
doi:

Substances chimiques

Lipopolysaccharides 0
Tumor Necrosis Factor-alpha 0
Berberine 0I8Y3P32UF
Phosphofructokinase-2 EC 2.7.1.105
Glucose IY9XDZ35W2

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2309-2318

Informations de copyright

Copyright © 2022 by The American Association of Immunologists, Inc.

Auteurs

Min Li (M)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Haifeng Zhang (H)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Yameng Zhang (Y)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Jinting Fan (J)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Jinhao Zhu (J)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Xinyue Gu (X)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Zhonghui Li (Z)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Chengyang Zhang (C)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Zhen Liu (Z)

College of Chemistry and Molecular Engineering, Zhengzhou University, Zhengzhou, China.

Yuebai Li (Y)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Mingxi Zang (M)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Ge Jin (G)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and.

Guoping Li (G)

College of Chemistry and Molecular Engineering, Zhengzhou University, Zhengzhou, China.

Yang Mi (Y)

Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou, China; and miyang@zzu.edu.cn.

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