Immunohistochemical determination of mismatch repair gene product in colorectal carcinomas in a young indigenous African cohort.


Journal

South African journal of surgery. Suid-Afrikaanse tydskrif vir chirurgie
ISSN: 2078-5151
Titre abrégé: S Afr J Surg
Pays: South Africa
ID NLM: 2984854R

Informations de publication

Date de publication:
Mar 2022
Historique:
entrez: 22 4 2022
pubmed: 23 4 2022
medline: 26 4 2022
Statut: ppublish

Résumé

Colorectal cancer (CRC) in the indigenous African population of South Africa is uncommon (age standardised incidence rates of 11.29 for males and 7.27/100 000 for females) and tends to occur at a young age. Lynch syndrome (LS), an inherited mismatch repair (MMR) gene abnormality, accounts for 3-4% of newly diagnosed CRCs in high incidence areas. There is some evidence that the contribution of an MMR abnormality to the overall CRC burden may be increased in low incidence areas. We aimed to determine the prevalence of MMR deficiency in an indigenous African population. A cohort of 66 self-declared indigenous African patients, less than 50 years of age at diagnosis with CRC was identified from clinical and pathological records. The original histopathology was reviewed to confirm the diagnosis and features suggestive of MMR abnormality determined (pushing edge, mucinous, lymphocytic infiltration, Crohn's like reaction). Where sufficient tissue was available, samples were sectioned and stained for the four MMR proteins. Histopathological examination confirmed adenocarcinoma in 31 individuals. At least one feature suggestive of MMR was identified in 22 of these specimens. Twenty-seven cases were stained for all four MMR proteins using standard immunohistochemistry (IHC). MMR deficiency was found in 37% ( MMR deficiency was common in colorectal carcinomas in the older patients in this cohort, but very young indigenous Africans CRCs do not appear to result from mismatch repair gene mutations.

Sections du résumé

BACKGROUND BACKGROUND
Colorectal cancer (CRC) in the indigenous African population of South Africa is uncommon (age standardised incidence rates of 11.29 for males and 7.27/100 000 for females) and tends to occur at a young age. Lynch syndrome (LS), an inherited mismatch repair (MMR) gene abnormality, accounts for 3-4% of newly diagnosed CRCs in high incidence areas. There is some evidence that the contribution of an MMR abnormality to the overall CRC burden may be increased in low incidence areas. We aimed to determine the prevalence of MMR deficiency in an indigenous African population.
METHODS METHODS
A cohort of 66 self-declared indigenous African patients, less than 50 years of age at diagnosis with CRC was identified from clinical and pathological records. The original histopathology was reviewed to confirm the diagnosis and features suggestive of MMR abnormality determined (pushing edge, mucinous, lymphocytic infiltration, Crohn's like reaction). Where sufficient tissue was available, samples were sectioned and stained for the four MMR proteins.
RESULTS RESULTS
Histopathological examination confirmed adenocarcinoma in 31 individuals. At least one feature suggestive of MMR was identified in 22 of these specimens. Twenty-seven cases were stained for all four MMR proteins using standard immunohistochemistry (IHC). MMR deficiency was found in 37% (
CONCLUSION CONCLUSIONS
MMR deficiency was common in colorectal carcinomas in the older patients in this cohort, but very young indigenous Africans CRCs do not appear to result from mismatch repair gene mutations.

Identifiants

pubmed: 35451266

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

28-33

Informations de copyright

Copyright© Authors.

Auteurs

R Holla (R)

Department of Gastroenterology, Vrije Universiteit Amsterdam, The Netherlands.

A Vorster (A)

MRC Human Genetics Research Unit, Institute of Infectious Diseases and Molecular Medicine, University of Cape Town and Affiliated Hospitals, South Africa.

M Locketz (M)

Division of Anatomical Pathology, National Health Laboratory Service and the University of Cape Town, South Africa.

M De Haas (M)

MRC Human Genetics Research Unit, Institute of Infectious Diseases and Molecular Medicine, University of Cape Town and Affiliated Hospitals, South Africa.

O A Oke (OA)

Colorectal Surgery Unit, Groote Schuur Hospital, University of Cape Town, South Africa.

D Govender (D)

Division of Anatomical Pathology, National Health Laboratory Service and the University of Cape Town, South Africa.

R Ramesar (R)

MRC Human Genetics Research Unit, Institute of Infectious Diseases and Molecular Medicine, University of Cape Town and Affiliated Hospitals, South Africa.

P A Goldberg (PA)

Colorectal Surgery Unit, Groote Schuur Hospital, University of Cape Town, South Africa.

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