Immunometabolism-fit: How exercise and training can modify T cell and macrophage metabolism in health and disease.


Journal

Exercise immunology review
ISSN: 1077-5552
Titre abrégé: Exerc Immunol Rev
Pays: Germany
ID NLM: 9505535

Informations de publication

Date de publication:
2022
Historique:
entrez: 22 4 2022
pubmed: 23 4 2022
medline: 27 4 2022
Statut: ppublish

Résumé

The term immunometabolism describes cellular and molecular metabolic processes that control the immune system and the associated immune responses. Acute exercise and regular physical activity have a substantial influence on the metabolism and the immune system, so that both processes are closely associated and influence each other bidirectionally. We limit the review here to focus on metabolic phenotypes and metabolic plasticity of T cells and macrophages to describe the complex role of acute exercise stress and regular physical activity on these cell types. The metabolic and immunological consequences of the social problem of inactivity and how, conversely, an active lifestyle can break this vicious circle, are then described. Finally, these aspects are evaluated against the background of an aging society. T cells and macrophages show high sensitivity to changes in their metabolic environment, which indirectly or directly affects their central functions. Physical activity and sedentary behaviour have an important influence on metabolic status, thereby modifying immune cell phenotypes and influencing immunological plasticity. A detailed understanding of the interactions between acute and chronic physical activity, sedentary behaviour, and the metabolic status of immune cells, can help to target the dysregulated immune system of people who live in a much too inactive society.

Sections du résumé

BACKGROUND BACKGROUND
The term immunometabolism describes cellular and molecular metabolic processes that control the immune system and the associated immune responses. Acute exercise and regular physical activity have a substantial influence on the metabolism and the immune system, so that both processes are closely associated and influence each other bidirectionally.
SCOPE OF REVIEW METHODS
We limit the review here to focus on metabolic phenotypes and metabolic plasticity of T cells and macrophages to describe the complex role of acute exercise stress and regular physical activity on these cell types. The metabolic and immunological consequences of the social problem of inactivity and how, conversely, an active lifestyle can break this vicious circle, are then described. Finally, these aspects are evaluated against the background of an aging society.
MAJOR CONCLUSIONS CONCLUSIONS
T cells and macrophages show high sensitivity to changes in their metabolic environment, which indirectly or directly affects their central functions. Physical activity and sedentary behaviour have an important influence on metabolic status, thereby modifying immune cell phenotypes and influencing immunological plasticity. A detailed understanding of the interactions between acute and chronic physical activity, sedentary behaviour, and the metabolic status of immune cells, can help to target the dysregulated immune system of people who live in a much too inactive society.

Identifiants

pubmed: 35452394

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

29-46

Informations de copyright

Copyright © 2022 International Society of Exercise and Immunology. All rights reserved.

Auteurs

José Cesar Rosa-Neto (JC)

Immunometabolism Research Group, Department of Cell Biology and Development, Institute of Biomedical Sciences 1, University of São Paulo (ICB1-USP), São Paulo 05508-000, Brazil.

Fábio Santos Lira (FS)

Exercise and Immunometabolism Research Group, Department of Physical Education, São Paulo State University (UNESP), Presidente Prudente 19060-900, Brazil.

Jonathan Peter Little (JP)

School of Health and Exercise Sciences, University of British Columbia, Okanagan Campus, Kelowna, British Columbia, CANADA.

Graham Landells (G)

School of Health and Exercise Sciences, University of British Columbia, Okanagan Campus, Kelowna, British Columbia, CANADA.

Hashim Islam (H)

School of Health and Exercise Sciences, University of British Columbia, Okanagan Campus, Kelowna, British Columbia, CANADA.

Bénédicte Chazaud (B)

Institut NeuroMyoGene, Unité Physiopathologie et Génétique du Neurone et du Muscle, Univ Lyon, CNRS 5261, INSERM U1315, Université Claude Bernard Lyon 1, Lyon, France.

David B Pyne (DB)

Research Institute for Sport and Exercise, University of Canberra, Canberra, ACT 2617, Australia.

Ana Maria Teixeira (AM)

University of Coimbra, Research Center for Sports and Physical Activity, Faculty of Sports Science and Physical Education, Coimbra, Portugal.

Helena Batatinha (H)

Immunometabolism Research Group, Department of Cell Biology and Development, Institute of Biomedical Sciences 1, University of São Paulo (ICB1-USP), São Paulo 05508-000, Brazil.

Barbara Moura Antunes (B)

Exercise and Immunometabolism Research Group, Department of Physical Education, São Paulo State University (UNESP), Presidente Prudente 19060-900, Brazil.
Facultad de Deportes Campus Ensenada, Universidad Autónoma de Baja California, México.

Luciele Guerra Minuzzi (L)

Exercise and Immunometabolism Research Group, Department of Physical Education, São Paulo State University (UNESP), Presidente Prudente 19060-900, Brazil.
University of Coimbra, Research Center for Sports and Physical Activity, Faculty of Sports Science and Physical Education, Coimbra, Portugal.

Jana Palmowski (J)

Department of Exercise Physiology and Sports Therapy, Institute of Sports Science, Justus Liebig University Giessen, 35394 Giessen, Germany.

Richard J Simpson (RJ)

School of Nutritional Sciences and Wellness; Department of Pediatrics; Department of Immunobiology, The University of Arizona, Tucson, AZ, USA.

Karsten Krüger (K)

Department of Exercise Physiology and Sports Therapy, Institute of Sports Science, Justus Liebig University Giessen, 35394 Giessen, Germany.

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Classifications MeSH