Muscle Damage in Dystrophic mdx Mice Is Influenced by the Activity of Ca
Duchenne muscular dystrophy
KCa3.1
Kcnn4
fiber size
fibroblasts
fibrosis
grip strength
hanging time
macrophages
neuromuscular junction
Journal
Life (Basel, Switzerland)
ISSN: 2075-1729
Titre abrégé: Life (Basel)
Pays: Switzerland
ID NLM: 101580444
Informations de publication
Date de publication:
05 Apr 2022
05 Apr 2022
Historique:
received:
28
02
2022
revised:
30
03
2022
accepted:
31
03
2022
entrez:
23
4
2022
pubmed:
24
4
2022
medline:
24
4
2022
Statut:
epublish
Résumé
Duchenne muscular dystrophy (DMD) is an X-linked disease, caused by a mutant dystrophin gene, leading to muscle membrane instability, followed by muscle inflammation, infiltration of pro-inflammatory macrophages and fibrosis. The calcium-activated potassium channel type 3.1 (K
Identifiants
pubmed: 35455028
pii: life12040538
doi: 10.3390/life12040538
pmc: PMC9025295
pii:
doi:
Types de publication
Journal Article
Langues
eng
Subventions
Organisme : Sapienza University of Rome
ID : RP1201727EA43399
Organisme : Ministry of Education, Universities and Research
ID : PRIN20178L7WRS
Organisme : Ministry of Education, Universities and Research
ID : PRIN2020Z73J5A
Organisme : Italian Association for Cancer Research
ID : IG-23010
Organisme : Duchenne Parent Project Netherlands
ID : small grant proposal ref. 19.006
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