Proton Pump Inhibitors Ameliorate Capecitabine-induced Hand-Foot Syndrome in Patients With Breast Cancer: A Retrospective Study.


Journal

Anticancer research
ISSN: 1791-7530
Titre abrégé: Anticancer Res
Pays: Greece
ID NLM: 8102988

Informations de publication

Date de publication:
May 2022
Historique:
received: 25 02 2022
revised: 18 03 2022
accepted: 23 03 2022
entrez: 30 4 2022
pubmed: 1 5 2022
medline: 4 5 2022
Statut: ppublish

Résumé

Hand-foot syndrome (HFS) is the most common adverse event associated with capecitabine, and its pathogenesis is known to be associated with inflammation. Proton pump inhibitors (PPIs) reportedly exert anti-inflammatory effects; however, the impact of PPIs on capecitabine-induced HFS needs to be clarified in the clinical setting. In the present study, we retrospectively investigated the efficacy and safety of PPIs in patients with breast cancer receiving capecitabine. We analyzed the effects of PPIs on the development of severe HFS (grade ≥2), progression-free survival (PFS), and overall survival (OS) in 195 patients who received capecitabine chemotherapy for breast cancer. In total, 50 patients (26%) were treated with PPIs, while 145 patients (74%) did not receive PPIs. The incidence of severe HFS was significantly lower in patients who received PPIs (18%) than in patients who did not receive PPIs (43%, p=0.001), and the discontinuation rate of capecitabine therapy due to HFS was also lower in patients receiving PPIs than in those who did not receive PPIs (p=0.003). Multivariate analysis revealed that concomitant PPIs use was an independent factor that significantly contributed to the prevention of severe HFS (odds ratio (OR)=0.265, p=0.003). Meanwhile, no significant difference in median PFS and OS values was observed between patients treated with and without PPIs. Concomitant use of PPIs could ameliorate capecitabine-induced HFS in patients with breast cancer.

Sections du résumé

BACKGROUND/AIM OBJECTIVE
Hand-foot syndrome (HFS) is the most common adverse event associated with capecitabine, and its pathogenesis is known to be associated with inflammation. Proton pump inhibitors (PPIs) reportedly exert anti-inflammatory effects; however, the impact of PPIs on capecitabine-induced HFS needs to be clarified in the clinical setting. In the present study, we retrospectively investigated the efficacy and safety of PPIs in patients with breast cancer receiving capecitabine.
PATIENTS AND METHODS METHODS
We analyzed the effects of PPIs on the development of severe HFS (grade ≥2), progression-free survival (PFS), and overall survival (OS) in 195 patients who received capecitabine chemotherapy for breast cancer.
RESULTS RESULTS
In total, 50 patients (26%) were treated with PPIs, while 145 patients (74%) did not receive PPIs. The incidence of severe HFS was significantly lower in patients who received PPIs (18%) than in patients who did not receive PPIs (43%, p=0.001), and the discontinuation rate of capecitabine therapy due to HFS was also lower in patients receiving PPIs than in those who did not receive PPIs (p=0.003). Multivariate analysis revealed that concomitant PPIs use was an independent factor that significantly contributed to the prevention of severe HFS (odds ratio (OR)=0.265, p=0.003). Meanwhile, no significant difference in median PFS and OS values was observed between patients treated with and without PPIs.
CONCLUSION CONCLUSIONS
Concomitant use of PPIs could ameliorate capecitabine-induced HFS in patients with breast cancer.

Identifiants

pubmed: 35489750
pii: 42/5/2591
doi: 10.21873/anticanres.15737
doi:

Substances chimiques

Proton Pump Inhibitors 0
Capecitabine 6804DJ8Z9U

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2591-2598

Informations de copyright

Copyright © 2022 International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Auteurs

Miho Takemura (M)

Department of Clinical Pharmacy Research and Education, Graduate School of Pharmaceutical Sciences, Osaka University, Suita, Japan.

Kenji Ikemura (K)

Department of Pharmacy, Osaka University Hospital, Suita, Japan; ikemurak@hp-drug.med.osaka-u.ac.jp.
Department of Hospital Pharmacy, Graduate School of Pharmaceutical Sciences, Osaka University, Suita, Japan.

Tetsuhiro Yoshinami (T)

Department of Breast and Endocrine Surgery, Graduate School of Medicine, Osaka University, Suita, Japan.

Yuji Toyozumi (Y)

Department of Hospital Pharmacy, Graduate School of Pharmaceutical Sciences, Osaka University, Suita, Japan.

Takuya Shintani (T)

Department of Pharmacy, Osaka University Hospital, Suita, Japan.

Mikiko Ueda (M)

Department of Clinical Pharmacy Research and Education, Graduate School of Pharmaceutical Sciences, Osaka University, Suita, Japan.

Kenzo Shimazu (K)

Department of Breast and Endocrine Surgery, Graduate School of Medicine, Osaka University, Suita, Japan.

Masahiro Okuda (M)

Department of Pharmacy, Osaka University Hospital, Suita, Japan.
Department of Hospital Pharmacy, Graduate School of Pharmaceutical Sciences, Osaka University, Suita, Japan.

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Classifications MeSH