Differences in saliva ACE2 activity among infected and non-infected adult and pediatric population exposed to SARS-CoV-2.


Journal

The Journal of infection
ISSN: 1532-2742
Titre abrégé: J Infect
Pays: England
ID NLM: 7908424

Informations de publication

Date de publication:
07 2022
Historique:
received: 28 09 2021
revised: 02 02 2022
accepted: 25 04 2022
pubmed: 2 5 2022
medline: 15 6 2022
entrez: 1 5 2022
Statut: ppublish

Résumé

Variations in the ACE2 activity in saliva could explain the striking differences of susceptibility to infection and risk of severe disease. We analyze the activity of ACE2 in saliva in different population groups across a wide age range and disease status during April to June 2020, before SARS-CoV-2 vaccine implementation, and we establish differences between infected people and participants considered resistant (highly exposed healthcare workers and children who cohabited with parents with COVID-19 without isolation and remain IgG negative). We included 74 adults, of which 47 (64%) were susceptible and 27 (36%) were resistant, and 79 children, of which 41 (52%) were susceptible and 38 (48%) were resistant. Resistant adults have significantly lower ACE2 activity in saliva than susceptible adults and non-significant higher values than susceptible and resistant children. ACE2 activity is similar in the susceptible and resistant pediatric population (p = 0.527). In contrast, we observe an increase in activity as the disease's severity increases among the adult population (mild disease vs. severe disease, 39 vs. 105 FU, p = 0.039; severe disease vs. resistant, 105 vs. 31 FU, p < 0.001). using an enzymatic test, we show that ACE2 activity in saliva correlates with the susceptibility to SARS-Cov-2 infection and disease severity. Children and adults with low-susceptibility to SARS-Cov-2 infection showed the lowest ACE2 activity. These findings could inform future strategies to identify at-risk individuals.

Sections du résumé

BACKGROUND
Variations in the ACE2 activity in saliva could explain the striking differences of susceptibility to infection and risk of severe disease.
METHODS
We analyze the activity of ACE2 in saliva in different population groups across a wide age range and disease status during April to June 2020, before SARS-CoV-2 vaccine implementation, and we establish differences between infected people and participants considered resistant (highly exposed healthcare workers and children who cohabited with parents with COVID-19 without isolation and remain IgG negative).
RESULTS
We included 74 adults, of which 47 (64%) were susceptible and 27 (36%) were resistant, and 79 children, of which 41 (52%) were susceptible and 38 (48%) were resistant. Resistant adults have significantly lower ACE2 activity in saliva than susceptible adults and non-significant higher values than susceptible and resistant children. ACE2 activity is similar in the susceptible and resistant pediatric population (p = 0.527). In contrast, we observe an increase in activity as the disease's severity increases among the adult population (mild disease vs. severe disease, 39 vs. 105 FU, p = 0.039; severe disease vs. resistant, 105 vs. 31 FU, p < 0.001).
CONCLUSIONS
using an enzymatic test, we show that ACE2 activity in saliva correlates with the susceptibility to SARS-Cov-2 infection and disease severity. Children and adults with low-susceptibility to SARS-Cov-2 infection showed the lowest ACE2 activity. These findings could inform future strategies to identify at-risk individuals.

Identifiants

pubmed: 35490737
pii: S0163-4453(22)00248-1
doi: 10.1016/j.jinf.2022.04.041
pmc: PMC9050198
pii:
doi:

Substances chimiques

COVID-19 Vaccines 0
ACE2 protein, human EC 3.4.17.23
Angiotensin-Converting Enzyme 2 EC 3.4.17.23

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

86-89

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2022. Published by Elsevier Ltd.

Déclaration de conflit d'intérêts

Declaration of Competing Interest Outside the submitted work, S. S.-V. reports personal fees from ViiV Healthcare, Janssen Cilag, Gilead Sciences, and MSD as well as non-financial support from ViiV Healthcare and Gilead Sciences and research grants from MSD and Gilead Sciences. J.M.-S. reports non-financial support from ViiV Healthcare, Gilead Sciences, and Jannsen Cilag. S.M. reports personal fees and non-financial from ViiV Healthcare, Janssen, Gilead Sciences, and MSD, as well as grants from MSD, ViiV Healthcare, and Gilead Sciences.

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Auteurs

Daniel Jiménez (D)

Department of Infectious Diseases, Hospital Universitario Ramón y Cajal, IRYCIS, Universidad de Alcalá and CIBERINFEC, Madrid, Spain.

Javier Martínez-Sanz (J)

Department of Infectious Diseases, Hospital Universitario Ramón y Cajal, IRYCIS, Universidad de Alcalá and CIBERINFEC, Madrid, Spain. Electronic address: javier.martinez.sanz@salud.madrid.org.

Talía Sainz (T)

Department of Pediatrics, Tropical and Infectious Diseases, Hospital La Paz, and La PazResearch Institute (IdiPAZ), Traslational Research Network of Pediatric Infectious Diseases (RITIP), and CIBERINFEC, Madrid, Spain.

Cristina Calvo (C)

Department of Pediatrics, Tropical and Infectious Diseases, Hospital La Paz, and La PazResearch Institute (IdiPAZ), Traslational Research Network of Pediatric Infectious Diseases (RITIP), and CIBERINFEC, Madrid, Spain.

Ana Méndez-Echevarría (A)

Department of Pediatrics, Tropical and Infectious Diseases, Hospital La Paz, and La PazResearch Institute (IdiPAZ), Traslational Research Network of Pediatric Infectious Diseases (RITIP), and CIBERINFEC, Madrid, Spain.

Elena Moreno (E)

Department of Infectious Diseases, Hospital Universitario Ramón y Cajal, IRYCIS, Universidad de Alcalá and CIBERINFEC, Madrid, Spain.

Daniel Blázquez-Gamero (D)

Pediatric Infectious Diseases Unit. Hospital Universitario 12 de Octubre. Universidad Complutense, Instituto de Investigación Hospital 12 de Octubre (imas12), Traslational Research Network of Pediatric Infectious Diseases (RITIP), Madrid, Spain.

Pilar Vizcarra (P)

Department of Infectious Diseases, Hospital Universitario Ramón y Cajal, IRYCIS, Universidad de Alcalá and CIBERINFEC, Madrid, Spain.

Mario Rodríguez (M)

Department of Microbiology, Facultad de Medicina, Hospital Universitario Ramón y Cajal, Universidad de Alcalá, IRYCIS, Madrid, Spain.

Robert Jenkins (R)

Reactomix S.L., Granada 18003, Spain.

Matilde Sánchez-Conde (M)

Department of Infectious Diseases, Hospital Universitario Ramón y Cajal, IRYCIS, Universidad de Alcalá and CIBERINFEC, Madrid, Spain.

Raquel Ron (R)

Department of Infectious Diseases, Hospital Universitario Ramón y Cajal, IRYCIS, Universidad de Alcalá and CIBERINFEC, Madrid, Spain.

Francesca Norman (F)

Department of Infectious Diseases, Hospital Universitario Ramón y Cajal, IRYCIS, Universidad de Alcalá and CIBERINFEC, Madrid, Spain.

Santiago Moreno (S)

Department of Infectious Diseases, Hospital Universitario Ramón y Cajal, IRYCIS, Universidad de Alcalá and CIBERINFEC, Madrid, Spain.

Manuel Ferrer (M)

Institute of Catalysis, Consejo Superior de Investigaciones Científicas (CSIC), Madrid, Spain.

Sergio Serrano-Villar (S)

Department of Infectious Diseases, Hospital Universitario Ramón y Cajal, IRYCIS, Universidad de Alcalá and CIBERINFEC, Madrid, Spain. Electronic address: sergio.serrano@salud.madrid.org.

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Classifications MeSH