Pulsatile pulmonary artery pressure in a large animal model of chronic thromboembolic pulmonary hypertension: Similarities and differences with human data.
animal models
pathophysiology
pulmonary hypertension experimental
right ventricle function and dysfunction
Journal
Pulmonary circulation
ISSN: 2045-8932
Titre abrégé: Pulm Circ
Pays: United States
ID NLM: 101557243
Informations de publication
Date de publication:
Jan 2022
Jan 2022
Historique:
received:
27
04
2021
revised:
27
10
2021
accepted:
20
11
2021
entrez:
4
5
2022
pubmed:
5
5
2022
medline:
5
5
2022
Statut:
epublish
Résumé
A striking feature of the human pulmonary circulation is that mean (mPAP) and systolic (sPAP) pulmonary artery pressures (PAPs) are strongly related and, thus, are essentially redundant. According to the empirical formula documented under normotensive and hypertensive conditions (mPAP = 0.61 sPAP + 2 mmHg), sPAP matches ~160%mPAP on average. This attests to the high pulsatility of PAP, as also witnessed by the near equality of PA pulse pressure and mPAP. Our prospective study tested if pressure redundancy and high pulsatility also apply in a piglet model of chronic thromboembolic pulmonary hypertension (CTEPH). At baseline (Week-0, W0), Sham (
Identifiants
pubmed: 35506099
doi: 10.1002/pul2.12017
pii: PUL212017
pmc: PMC9052967
doi:
Types de publication
Journal Article
Langues
eng
Pagination
e12017Informations de copyright
© 2021 The Authors. Pulmonary Circulation published by Wiley Periodicals LLC on behalf of the Pulmonary Vascular Research Institute.
Déclaration de conflit d'intérêts
The authors declare that there are no conflicts of interest in the field (physiology).
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