Sclerosing Paragangliomas: Correlations of Histological Features with Patients' Genotype and Vesicular Monoamine Transporter Expression.

Genotype Germ-line mutation Immunohistochemistry Paragangliomas Pheochromocytomas Sclerosis Vesicular monoamine transport proteins

Journal

Head and neck pathology
ISSN: 1936-0568
Titre abrégé: Head Neck Pathol
Pays: United States
ID NLM: 101304010

Informations de publication

Date de publication:
Dec 2022
Historique:
received: 29 12 2021
accepted: 13 04 2022
pubmed: 8 5 2022
medline: 15 12 2022
entrez: 7 5 2022
Statut: ppublish

Résumé

Paragangliomas and pheochromocytomas are rare neuroendocrine tumors, carrying a germ-line mutation in 40% patients. Sclerosis is a rare histological feature in these tumors. We investigated the possible correlations between histological findings, first sclerosis, immunoreactivity for vesicular catecholamine transporters (VMAT1/VMAT2) and patients' genotype in a consecutive series of 57 tumors (30 paragangliomas and 27 pheochromocytomas) from 55 patients. The M-GAPP grading system, sclerosis (0-3 scale) and VMAT1/VMAT2 (0-6 scale) immunoreactivity scores were assessed. Germ-line mutations of Succinate Dehydrogenase genes, RET proto-oncogene and Von Hippel Lindau tumor suppressor gene were searched. A germ-line mutation was found in 25/55 (45.5%) patients, mainly with paraganglioma (N = 14/30, 46,66%). Significant (score ≥ 2) tumor sclerosis was found in 9 (16.1%) tumors, i.e., 7 paragangliomas and 2 pheochromocytomas, most of them (8/9) from patients with a germ-line mutation. M-GAPP score was higher in the mutation status (in 76% of patients involving the SDHx genes, in 12% the RET gene and in the remaining 12% the VHL gene) and in tumors with sclerosis (p < 0.05). Spearman's rank correlation showed a strong correlation of germ-line mutations with M-GAPP (p < 0.0001) and sclerosis (p = 0.0027) scores; a significant correlation was also found between sclerosis and M-GAPP scores (p = 0.029). VMAT1 expression was higher in paragangliomas than in pheochromocytomas (p = 0.0006), the highest scores being more frequent in mutation-bearing patients' tumors (p < 0.01). VMAT2 was highly expressed in all but two negative tumors. Sclerosis and VMAT1 expression were higher in paragangliomas than in pheochromocytomas; tumor sclerosis, M-GAPP and VMAT1 scores were associated to germ-line mutations. Sclerosis might represent a histological marker of tumor susceptibility, prompting to genetic investigations in paragangliomas.

Identifiants

pubmed: 35524772
doi: 10.1007/s12105-022-01455-4
pii: 10.1007/s12105-022-01455-4
pmc: PMC9729524
doi:

Substances chimiques

Vesicular Monoamine Transport Proteins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

998-1011

Informations de copyright

© 2022. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.

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Auteurs

Angela Pucci (A)

Department of Histopathology, Pisa University Hospital and Pisa University (FB), Via Roma, 57, 56126, Pisa, Italy. angelapucci@libero.it.

Alessandra Bacca (A)

Department of Clinical and Experimental Medicine, Pisa University Hospital and Pisa University (FB), Pisa, Italy.

Ivana Barravecchia (I)

Institute of Life Sciences, Scuola Superiore Sant'Anna, Pisa, Italy.

Iosè Di Stefano (I)

Department of Histopathology, Pisa University Hospital and Pisa University (FB), Via Roma, 57, 56126, Pisa, Italy.

Beatrice Belgio (B)

Department of Histopathology, Pisa University Hospital and Pisa University (FB), Via Roma, 57, 56126, Pisa, Italy.

Daniele Lorenzini (D)

Department of Histopathology, Pisa University Hospital and Pisa University (FB), Via Roma, 57, 56126, Pisa, Italy.
INT (National Institute of Tumors), Milan, Italy.

Liborio Torregrossa (L)

Department of Histopathology, Pisa University Hospital and Pisa University (FB), Via Roma, 57, 56126, Pisa, Italy.

Serena Chiacchio (S)

Department of Diagnostic, Interventional and Vascular Radiology and of Nuclear Medicine, Pisa University Hospital and Pisa University, Pisa, Italy.

Caterina Congregati (C)

Department of Translational Research and New Medical Technology, Pisa University Hospital and Pisa University, Pisa, Italy.

Gabriele Materazzi (G)

Department of Surgical, Molecular and Clinical Pathology and Critical Care, Pisa University Hospital and Pisa University, Pisa, Italy.

Mauro Ferrari (M)

Department of Cardiovascular Surgery, Pisa University Hospital and Pisa University, Pisa, Italy.

Debora Angeloni (D)

Institute of Life Sciences, Scuola Superiore Sant'Anna, Pisa, Italy.

Giampaolo Bernini (G)

Department of Clinical and Experimental Medicine, Pisa University Hospital and Pisa University (FB), Pisa, Italy.

Fulvio Basolo (F)

Department of Histopathology, Pisa University Hospital and Pisa University (FB), Via Roma, 57, 56126, Pisa, Italy.

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Classifications MeSH