Genetic and compound screens uncover factors modulating cancer cell response to indisulam.


Journal

Life science alliance
ISSN: 2575-1077
Titre abrégé: Life Sci Alliance
Pays: United States
ID NLM: 101728869

Informations de publication

Date de publication:
09 2022
Historique:
received: 22 12 2021
revised: 26 04 2022
accepted: 26 04 2022
entrez: 9 5 2022
pubmed: 10 5 2022
medline: 12 5 2022
Statut: epublish

Résumé

Discovering biomarkers of drug response and finding powerful drug combinations can support the reuse of previously abandoned cancer drugs in the clinic. Indisulam is an abandoned drug that acts as a molecular glue, inducing degradation of splicing factor RBM39 through interaction with CRL4

Identifiants

pubmed: 35534224
pii: 5/9/e202101348
doi: 10.26508/lsa.202101348
pmc: PMC9095732
pii:
doi:

Substances chimiques

Antineoplastic Agents 0
Intracellular Signaling Peptides and Proteins 0
N-(3-chloro-7-indolyl)-1,4-benzenedisulphonamide 0
RNA Splicing Factors 0
Sulfonamides 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2022 Pogacar et al.

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Auteurs

Ziva Pogacar (Z)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Kelvin Groot (K)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Fleur Jochems (F)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Matheus Dos Santos Dias (M)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Antonio Mulero-Sánchez (A)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Ben Morris (B)

The Netherlands Cancer Institute Robotics and Screening Center, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Mieke Roosen (M)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Leyma Wardak (L)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Giulia De Conti (G)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Arno Velds (A)

Genomics Core Facility, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Cor Lieftink (C)

The Netherlands Cancer Institute Robotics and Screening Center, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Bram Thijssen (B)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

Roderick L Beijersbergen (RL)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
The Netherlands Cancer Institute Robotics and Screening Center, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
Genomics Core Facility, The Netherlands Cancer Institute, Amsterdam, The Netherlands.

René Bernards (R)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands r.bernards@nki.nl r.ld.oliveira@amsterdamumc.nl.

Rodrigo Leite de Oliveira (R)

Division of Molecular Carcinogenesis, Oncode Institute, The Netherlands Cancer Institute, Amsterdam, The Netherlands r.bernards@nki.nl r.ld.oliveira@amsterdamumc.nl.

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