Disinfection By-Products in Drinking Water and Bladder Cancer: Evaluation of Risk Modification by Common Genetic Polymorphisms in Two Case-Control Studies.


Journal

Environmental health perspectives
ISSN: 1552-9924
Titre abrégé: Environ Health Perspect
Pays: United States
ID NLM: 0330411

Informations de publication

Date de publication:
05 2022
Historique:
entrez: 10 5 2022
pubmed: 11 5 2022
medline: 14 5 2022
Statut: ppublish

Résumé

By-products are formed when disinfectants react with organic matter in source water. The most common class of disinfection by-products, trihalomethanes (THMs), have been linked to bladder cancer. Several studies have shown exposure-response associations with THMs in drinking water and bladder cancer risk. Few epidemiologic studies have evaluated gene-environment interactions for total THMs (TTHMs) with known bladder cancer susceptibility variants. In this study, we investigated the combined effect on bladder cancer risk contributed by TTHMs, bladder cancer susceptibility variants identified through genome-wide association studies, and variants in several candidate genes. We analyzed data from two large case-control studies-the New England Bladder Cancer Study ( Of the 16 loci showing strong evidence of association with bladder cancer, rs907611 at 11p15.5 [leukocyte-specific protein 1 ( We found novel suggestive evidence for a multiplicative interaction between a putative bladder carcinogen, TTHMs, and genotypes of rs907611. Given the ubiquitous exposure to THMs, further work is needed to replicate and extend this finding and to understand potential molecular mechanisms. https://doi.org/10.1289/EHP9895.

Sections du résumé

BACKGROUND
By-products are formed when disinfectants react with organic matter in source water. The most common class of disinfection by-products, trihalomethanes (THMs), have been linked to bladder cancer. Several studies have shown exposure-response associations with THMs in drinking water and bladder cancer risk. Few epidemiologic studies have evaluated gene-environment interactions for total THMs (TTHMs) with known bladder cancer susceptibility variants.
OBJECTIVES
In this study, we investigated the combined effect on bladder cancer risk contributed by TTHMs, bladder cancer susceptibility variants identified through genome-wide association studies, and variants in several candidate genes.
METHODS
We analyzed data from two large case-control studies-the New England Bladder Cancer Study (
RESULTS
Of the 16 loci showing strong evidence of association with bladder cancer, rs907611 at 11p15.5 [leukocyte-specific protein 1 (
DISCUSSION
We found novel suggestive evidence for a multiplicative interaction between a putative bladder carcinogen, TTHMs, and genotypes of rs907611. Given the ubiquitous exposure to THMs, further work is needed to replicate and extend this finding and to understand potential molecular mechanisms. https://doi.org/10.1289/EHP9895.

Identifiants

pubmed: 35536285
doi: 10.1289/EHP9895
pmc: PMC9088962
doi:

Substances chimiques

Disinfectants 0
Drinking Water 0
Trihalomethanes 0
Water Pollutants, Chemical 0

Types de publication

Journal Article Meta-Analysis

Langues

eng

Sous-ensembles de citation

IM

Pagination

57006

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Auteurs

Laura E Beane Freeman (LE)

Barcelona Institute for Global Health, Barcelona, Spain.

Manolis Kogevinas (M)

Barcelona Institute for Global Health, Barcelona, Spain.
CIBER Epidemiologia y Salud Pública, Madrid, Spain.
Universitat Pompeu Fabra (UPF), Barcelona, Spain.
Hospital del Mar Medical Research Institute, Barcelona, Spain.

Kenneth P Cantor (KP)

Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute (NCI), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Bethesda, Maryland, USA.

Cristina M Villanueva (CM)

Barcelona Institute for Global Health, Barcelona, Spain.
CIBER Epidemiologia y Salud Pública, Madrid, Spain.
Universitat Pompeu Fabra (UPF), Barcelona, Spain.
Hospital del Mar Medical Research Institute, Barcelona, Spain.

Ludmila Prokunina-Olsson (L)

Laboratory of Translational Genomics, Division of Cancer Epidemiology and Genetics, NCI, NIH, DHHS, Bethesda, Maryland, USA.

Oscar Florez-Vargas (O)

Laboratory of Translational Genomics, Division of Cancer Epidemiology and Genetics, NCI, NIH, DHHS, Bethesda, Maryland, USA.

Jonine D Figueroa (JD)

Usher Institute, University of Edinburgh, Edinburgh, UK.
Cancer Research UK Edinburgh Centre, University of Edinburgh, Edinburgh, UK.

Mary H Ward (MH)

Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute (NCI), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Bethesda, Maryland, USA.

Stella Koutros (S)

Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute (NCI), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Bethesda, Maryland, USA.

Dalsu Baris (D)

Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute (NCI), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Bethesda, Maryland, USA.

Montserrat Garcia-Closas (M)

Trans-Divisional Research Program, Division of Cancer Epidemiology and Genetics, NCI, NIH, DHHS, Bethesda, Maryland, USA.

Molly Schwenn (M)

Maine Cancer Registry, Augusta, Maine, USA.

Allison Johnson (A)

Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute (NCI), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Bethesda, Maryland, USA.

Consol Serra (C)

Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute (NCI), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Bethesda, Maryland, USA.
Barcelona Institute for Global Health, Barcelona, Spain.
CIBER Epidemiologia y Salud Pública, Madrid, Spain.
Hospital del Mar Medical Research Institute, Barcelona, Spain.

Adonina Tardon (A)

Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute (NCI), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Bethesda, Maryland, USA.
CIBER Epidemiologia y Salud Pública, Madrid, Spain.

Reina Garcia-Closas (R)

Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute (NCI), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Bethesda, Maryland, USA.
Universitat Pompeu Fabra (UPF), Barcelona, Spain.

Alfredo Carrato (A)

Medical Oncology Department, Ramón y Cajal University Hospital, Madrid, Spain.
Alcalá University, Madrid, Spain.
Instituto Ramón y Cajal de Investigación Sanitaria, Madrid, Spain.
Centro de Investigación Biomédica en Red Cáncer, Madrid, Spain.

Nuria Malats (N)

Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute (NCI), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Bethesda, Maryland, USA.
Cancer Research UK Edinburgh Centre, University of Edinburgh, Edinburgh, UK.
Trans-Divisional Research Program, Division of Cancer Epidemiology and Genetics, NCI, NIH, DHHS, Bethesda, Maryland, USA.

Margaret R Karagas (MR)

Department of Epidemiology, Geisel School of Medicine at Dartmouth, Hanover, New Hampshire, USA.

Nathaniel Rothman (N)

Barcelona Institute for Global Health, Barcelona, Spain.

Debra T Silverman (DT)

Barcelona Institute for Global Health, Barcelona, Spain.

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