Genome-wide association of polygenic risk extremes for Alzheimer's disease in the UK Biobank.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
19 05 2022
Historique:
received: 03 12 2021
accepted: 10 05 2022
entrez: 19 5 2022
pubmed: 20 5 2022
medline: 24 5 2022
Statut: epublish

Résumé

In just over a decade, advances in genome-wide association studies (GWAS) have offered an approach to stratify individuals based on genetic risk for disease. Using recent Alzheimer's disease (AD) GWAS results as the base data, we determined each individual's polygenic risk score (PRS) in the UK Biobank dataset. Using individuals within the extreme risk distribution, we performed a GWAS that is agnostic of AD phenotype and is instead based on known genetic risk for disease. To interpret the functions of the new risk factors, we conducted phenotype analyses, including a phenome-wide association study. We identified 246 loci surpassing the significance threshold of which 229 were not reported in the base AD GWAS. These include loci that showed suggestive levels of association in the base GWAS and loci not previously suspected to be associated with AD. Among these, there are loci, such as IL34 and KANSL1, that have since been shown to be associated with AD in recent studies. We also show highly significant genetic correlations with multiple health-related outcomes that provide insights into prodromal symptoms and comorbidities. This is the first study to utilize PRS as a phenotype-agnostic group classification in AD genetic studies. We identify potential new loci for AD and detail phenotypic analysis of these PRS extremes.

Identifiants

pubmed: 35589863
doi: 10.1038/s41598-022-12391-2
pii: 10.1038/s41598-022-12391-2
pmc: PMC9120074
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

8404

Subventions

Organisme : Medical Research Council
ID : MC_PC_17228
Pays : United Kingdom
Organisme : Medical Research Council
ID : MC_QA137853
Pays : United Kingdom
Organisme : NIA NIH HHS
ID : R01 AG067426
Pays : United States
Organisme : NIH HHS
ID : R01AG067426
Pays : United States

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2022. The Author(s).

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Auteurs

Catarina Gouveia (C)

Department of Neurodegenerative Science, Van Andel Research Institute, 333 Bostwick Ave. N.E., Grand Rapids, MI, 49503-2518, USA.

Elizabeth Gibbons (E)

Department of Neurodegenerative Science, Van Andel Research Institute, 333 Bostwick Ave. N.E., Grand Rapids, MI, 49503-2518, USA.

Nadia Dehghani (N)

Department of Neurodegenerative Science, Van Andel Research Institute, 333 Bostwick Ave. N.E., Grand Rapids, MI, 49503-2518, USA.

James Eapen (J)

Department of Neurodegenerative Science, Van Andel Research Institute, 333 Bostwick Ave. N.E., Grand Rapids, MI, 49503-2518, USA.

Rita Guerreiro (R)

Department of Neurodegenerative Science, Van Andel Research Institute, 333 Bostwick Ave. N.E., Grand Rapids, MI, 49503-2518, USA.
Division of Psychiatry and Behavioral Medicine, Michigan State University College of Human Medicine, Grand Rapids, MI, USA.

Jose Bras (J)

Department of Neurodegenerative Science, Van Andel Research Institute, 333 Bostwick Ave. N.E., Grand Rapids, MI, 49503-2518, USA. Jose.Bras@vai.org.
Division of Psychiatry and Behavioral Medicine, Michigan State University College of Human Medicine, Grand Rapids, MI, USA. Jose.Bras@vai.org.

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