Enhancing Radioiodine Incorporation into Radioiodine-Refractory Thyroid Cancer with MAPK Inhibition (ERRITI): A Single-Center Prospective Two-Arm Study.


Journal

Clinical cancer research : an official journal of the American Association for Cancer Research
ISSN: 1557-3265
Titre abrégé: Clin Cancer Res
Pays: United States
ID NLM: 9502500

Informations de publication

Date de publication:
03 10 2022
Historique:
received: 09 02 2022
revised: 04 04 2022
accepted: 17 05 2022
pubmed: 21 5 2022
medline: 5 10 2022
entrez: 20 5 2022
Statut: ppublish

Résumé

Restoration of iodine incorporation (redifferentiation) by MAPK inhibition was achieved in previously radioiodine-refractory, unresectable thyroid carcinoma (RR-TC). However, results were unsatisfactory in BRAFV600E-mutant (BRAF-MUT) RR-TC. Here we assess safety and efficacy of redifferentiation therapy through genotype-guided MAPK-modulation in patients with BRAF-MUT or wildtype (BRAF-WT) RR-TC. In this prospective single-center, two-arm phase II study, patients received trametinib (BRAF-WT) or trametinib + dabrafenib (BRAF-MUT) for 21 ± 3 days. Redifferentiation was assessed by 123I-scintigraphy. In case of restored radioiodine uptake, 124I-guided 131I therapy was performed. Primary endpoint was the redifferentiation rate. Secondary endpoints were treatment response (thyroglobulin, RECIST 1.1) and safety. Parameters predicting successful redifferentiation were assessed using a receiver operating characteristic analysis and Youden J statistic. Redifferentiation was achieved in 7 of 20 (35%) patients, 2 of 6 (33%) in the BRAF-MUT and 5 of 14 (36%) in the BRAF-WT arm. Patients received a mean (range) activity of 300.0 (273.0-421.6) mCi for 131I therapy. Any thyroglobulin decline was seen in 57% (4/7) of the patients, RECIST 1.1 stable/partial response/progressive disease in 71% (5/7)/14% (1/7)/14% (1/7). Peak standardized uptake value (SUVpeak) < 10 on 2[18F]fluoro-2-deoxy-D-glucose (FDG)-PET was associated with successful redifferentiation (P = 0.01). Transient pyrexia (grade 3) and rash (grade 4) were noted in one patient each. Genotype-guided MAPK inhibition was safe and resulted in successful redifferentiation in about one third of patients in each arm. Subsequent 131I therapy led to a thyroglobulin (Tg) decline in more than half of the treated patients. Low tumor glycolytic rate as assessed by FDG-PET is predictive of redifferentiation success. See related commentary by Cabanillas et al., p. 4164.

Identifiants

pubmed: 35594174
pii: 699089
doi: 10.1158/1078-0432.CCR-22-0437
pmc: PMC9527501
doi:

Substances chimiques

Iodine Radioisotopes 0
Iodine-124 0
Iodine-131 0
Protein Kinase Inhibitors 0
Fluorodeoxyglucose F18 0Z5B2CJX4D
Thyroglobulin 9010-34-8
Proto-Oncogene Proteins B-raf EC 2.7.11.1

Types de publication

Editorial

Langues

eng

Sous-ensembles de citation

IM

Pagination

4194-4202

Commentaires et corrections

Type : CommentIn

Informations de copyright

©2022 The Authors; Published by the American Association for Cancer Research.

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Auteurs

Manuel Weber (M)

Clinic for Nuclear Medicine, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.

David Kersting (D)

Clinic for Nuclear Medicine, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.

Burkhard Riemann (B)

Department of Nuclear Medicine, University Hospital Münster, Münster, Germany.

Tim Brandenburg (T)

German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.
Department of Endocrinology and Metabolism, Division of Laboratory Research, University Hospital Essen, University Duisburg-Essen, Essen, Germany.

Dagmar Führer-Sakel (D)

German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.
Department of Endocrinology and Metabolism, Division of Laboratory Research, University Hospital Essen, University Duisburg-Essen, Essen, Germany.

Frank Grünwald (F)

Department of Nuclear Medicine, University Hospital Frankfurt, Frankfurt, Germany.

Michael C Kreissl (MC)

Clinic of Radiology and Nuclear Medicine, University Hospital Magdeburg, Magdeburg, Germany.

Henning Dralle (H)

Department of General, Visceral and Transplantation Surgery, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.

Frank Weber (F)

Department of General, Visceral and Transplantation Surgery, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.

Kurt Werner Schmid (KW)

Institute of Pathology, University Hospital Essen, University Duisburg-Essen, Essen, Germany.

Ken Herrmann (K)

Clinic for Nuclear Medicine, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.

Walter Jentzen (W)

Clinic for Nuclear Medicine, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.

Hong Grafe (H)

Clinic for Nuclear Medicine, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.

Christoph Rischpler (C)

Clinic for Nuclear Medicine, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.

Sarah Theurer (S)

Institute of Pathology, University Hospital Essen, University Duisburg-Essen, Essen, Germany.

Andreas Bockisch (A)

Clinic for Nuclear Medicine, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.

James Nagarajah (J)

Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, New York.
Department of Medical Imaging, Radboud University Medical Center, Nijmegen, Netherlands.

Wolfgang P Fendler (WP)

Clinic for Nuclear Medicine, University Hospital Essen, University Duisburg-Essen, Essen, Germany.
German Cancer Consortium (DKTK) partner site Essen, Essen, Germany.

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