Determining breast cancer biomarker status and associated morphological features using deep learning.

Breast cancer Pathology

Journal

Communications medicine
ISSN: 2730-664X
Titre abrégé: Commun Med (Lond)
Pays: England
ID NLM: 9918250414506676

Informations de publication

Date de publication:
2021
Historique:
received: 30 01 2021
accepted: 18 06 2021
entrez: 23 5 2022
pubmed: 24 5 2022
medline: 24 5 2022
Statut: epublish

Résumé

Breast cancer management depends on biomarkers including estrogen receptor, progesterone receptor, and human epidermal growth factor receptor 2 (ER/PR/HER2). Though existing scoring systems are widely used and well-validated, they can involve costly preparation and variable interpretation. Additionally, discordances between histology and expected biomarker findings can prompt repeat testing to address biological, interpretative, or technical reasons for unexpected results. We developed three independent deep learning systems (DLS) to directly predict ER/PR/HER2 status for both focal tissue regions (patches) and slides using hematoxylin-and-eosin-stained (H&E) images as input. Models were trained and evaluated using pathologist annotated slides from three data sources. Areas under the receiver operator characteristic curve (AUCs) were calculated for test sets at both a patch-level (>135 million patches, 181 slides) and slide-level ( The patch-level AUCs are 0.939 (95%CI 0.936-0.941), 0.938 (0.936-0.940), and 0.808 (0.802-0.813) for ER/PR/HER2, respectively. At the slide level, AUCs are 0.86 (95%CI 0.84-0.87), 0.75 (0.73-0.77), and 0.60 (0.56-0.64) for ER/PR/HER2, respectively. Interpretability analyses show known biomarker-histomorphology associations including associations of low-grade and lobular histology with ER/PR positivity, and increased inflammatory infiltrates with triple-negative staining. This study presents rapid breast cancer biomarker estimation from routine H&E slides and builds on prior advances by prioritizing interpretability of computationally learned features in the context of existing pathological knowledge.

Sections du résumé

Background UNASSIGNED
Breast cancer management depends on biomarkers including estrogen receptor, progesterone receptor, and human epidermal growth factor receptor 2 (ER/PR/HER2). Though existing scoring systems are widely used and well-validated, they can involve costly preparation and variable interpretation. Additionally, discordances between histology and expected biomarker findings can prompt repeat testing to address biological, interpretative, or technical reasons for unexpected results.
Methods UNASSIGNED
We developed three independent deep learning systems (DLS) to directly predict ER/PR/HER2 status for both focal tissue regions (patches) and slides using hematoxylin-and-eosin-stained (H&E) images as input. Models were trained and evaluated using pathologist annotated slides from three data sources. Areas under the receiver operator characteristic curve (AUCs) were calculated for test sets at both a patch-level (>135 million patches, 181 slides) and slide-level (
Results UNASSIGNED
The patch-level AUCs are 0.939 (95%CI 0.936-0.941), 0.938 (0.936-0.940), and 0.808 (0.802-0.813) for ER/PR/HER2, respectively. At the slide level, AUCs are 0.86 (95%CI 0.84-0.87), 0.75 (0.73-0.77), and 0.60 (0.56-0.64) for ER/PR/HER2, respectively. Interpretability analyses show known biomarker-histomorphology associations including associations of low-grade and lobular histology with ER/PR positivity, and increased inflammatory infiltrates with triple-negative staining.
Conclusions UNASSIGNED
This study presents rapid breast cancer biomarker estimation from routine H&E slides and builds on prior advances by prioritizing interpretability of computationally learned features in the context of existing pathological knowledge.

Identifiants

pubmed: 35602213
doi: 10.1038/s43856-021-00013-3
pii: 13
pmc: PMC9037318
doi:

Types de publication

Journal Article

Langues

eng

Pagination

14

Informations de copyright

© The Author(s) 2021.

Déclaration de conflit d'intérêts

Competing interestsThis study was funded by Google LLC and Verily Life Sciences. P.G., R.J., H.W., F.T., M.M., G.S.C., L.H.P., Y.L., C.H.M., D.F.S., and P.-H.C.C. are employees of Google LLC and own Alphabet stock. I.F.-A. and T.B. are consultants of Google LLC. M.T., D.J.D., E.A.R., P.R., N.O., J.H.W., and C.R. declare no competing interests.

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Auteurs

Paul Gamble (P)

Google Health, Palo Alto, CA USA.

Ronnachai Jaroensri (R)

Google Health, Palo Alto, CA USA.

Hongwu Wang (H)

Google Health, Palo Alto, CA USA.

Fraser Tan (F)

Google Health, Palo Alto, CA USA.

Melissa Moran (M)

Google Health, Palo Alto, CA USA.

Trissia Brown (T)

Google Health via Vituity, Emeryville, CA USA.

Isabelle Flament-Auvigne (I)

Google Health via Vituity, Emeryville, CA USA.

Emad A Rakha (EA)

Department of Pathology, School of Medicine, University of Nottingham, Nottingham, UK.

Michael Toss (M)

Department of Pathology, School of Medicine, University of Nottingham, Nottingham, UK.

David J Dabbs (DJ)

John A. Burns University of Hawaii Cancer Center, Honolulu, HI USA.
Department of Pathology, Magee-Womens Hospital of UPMC, Pittsburgh, PA USA.

Peter Regitnig (P)

Diagnostic and Research Institute of Pathology, Medical University of Graz, Graz, Austria.

Niels Olson (N)

Defense Innovation Unit, Mountain View, CA USA.

James H Wren (JH)

Henry M. Jackson Foundation, Bethesda, MD USA.

Carrie Robinson (C)

Laboratory Department, Naval Medical Center San Diego, San Diego, CA USA.

Greg S Corrado (GS)

Google Health, Palo Alto, CA USA.

Lily H Peng (LH)

Google Health, Palo Alto, CA USA.

Yun Liu (Y)

Google Health, Palo Alto, CA USA.

Craig H Mermel (CH)

Google Health, Palo Alto, CA USA.

David F Steiner (DF)

Google Health, Palo Alto, CA USA.

Classifications MeSH