Infarct location and cognitive change in patients after acute ischemic stroke: The ICONS study.


Journal

Journal of the neurological sciences
ISSN: 1878-5883
Titre abrégé: J Neurol Sci
Pays: Netherlands
ID NLM: 0375403

Informations de publication

Date de publication:
15 07 2022
Historique:
received: 01 11 2021
revised: 04 05 2022
accepted: 05 05 2022
pubmed: 27 5 2022
medline: 9 6 2022
entrez: 26 5 2022
Statut: ppublish

Résumé

Post stroke cognitive impairment is closely related to the quality of life. We aimed to evaluate the association between infarct location and cognitive change over time after acute ischemic stroke (AIS). Patients were selected from the Impairment of Cognition and Sleep after AIS or transient ischemic attack in Chinese patients (ICONS) study. Infarct location was assessed by brain magnetic resonance imaging. Cognition was screened at two weeks and 12 months by the Montreal Cognitive Assessment (MoCA). The primary outcome was the cognitive change at 12 months compared to two weeks. We tested the associations with cognitive change using logistic regression analysis. A total of 865 patients were enrolled. The mean age of the study participants was 59.67 ± 10.92 years, and the median National Institutes of Health Stroke Scale was 3 (1-5). In a fully adjusted model, thalamic infarction was significantly associated with cognitive decline after 12 months following an AIS (odds ratio [OR] 4.873, 95% confidence interval [CI] 1.634-14.534; p = 0.005), independent of stroke etiology (p > 0.05). Thalamic infarction increased the risk of worse cognitive performance, as screened by MoCA in relatively young patients with minor ischemic stroke at 12 months, suggesting the thalamus may be a critical structure in preserving cognition after stroke.

Sections du résumé

BACKGROUND AND PURPOSE
Post stroke cognitive impairment is closely related to the quality of life. We aimed to evaluate the association between infarct location and cognitive change over time after acute ischemic stroke (AIS).
METHODS
Patients were selected from the Impairment of Cognition and Sleep after AIS or transient ischemic attack in Chinese patients (ICONS) study. Infarct location was assessed by brain magnetic resonance imaging. Cognition was screened at two weeks and 12 months by the Montreal Cognitive Assessment (MoCA). The primary outcome was the cognitive change at 12 months compared to two weeks. We tested the associations with cognitive change using logistic regression analysis.
RESULTS
A total of 865 patients were enrolled. The mean age of the study participants was 59.67 ± 10.92 years, and the median National Institutes of Health Stroke Scale was 3 (1-5). In a fully adjusted model, thalamic infarction was significantly associated with cognitive decline after 12 months following an AIS (odds ratio [OR] 4.873, 95% confidence interval [CI] 1.634-14.534; p = 0.005), independent of stroke etiology (p > 0.05).
CONCLUSIONS
Thalamic infarction increased the risk of worse cognitive performance, as screened by MoCA in relatively young patients with minor ischemic stroke at 12 months, suggesting the thalamus may be a critical structure in preserving cognition after stroke.

Identifiants

pubmed: 35617844
pii: S0022-510X(22)00138-1
doi: 10.1016/j.jns.2022.120276
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

120276

Informations de copyright

Copyright © 2021. Published by Elsevier B.V.

Auteurs

Chen Zhang (C)

National Clinical Research Center for Neurological Diseases, Beijing Tiantan Hospital, Capital Medical University, Beijing, China; Department of Neurology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China.

Yue Wang (Y)

National Clinical Research Center for Neurological Diseases, Beijing Tiantan Hospital, Capital Medical University, Beijing, China; Department of Neurology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China.

Shiping Li (S)

National Clinical Research Center for Neurological Diseases, Beijing Tiantan Hospital, Capital Medical University, Beijing, China; Department of Neurology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China.

Yuesong Pan (Y)

National Clinical Research Center for Neurological Diseases, Beijing Tiantan Hospital, Capital Medical University, Beijing, China.

Mengxing Wang (M)

National Clinical Research Center for Neurological Diseases, Beijing Tiantan Hospital, Capital Medical University, Beijing, China.

Xiaoling Liao (X)

National Clinical Research Center for Neurological Diseases, Beijing Tiantan Hospital, Capital Medical University, Beijing, China; Department of Neurology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China.

Jiong Shi (J)

National Clinical Research Center for Neurological Diseases, Beijing Tiantan Hospital, Capital Medical University, Beijing, China; Department of Neurology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China; Advanced Innovation Center for Human Brain Protection, Capital Medical University, Beijing, China. Electronic address: jiongshi@ncrcnd.org.cn.

Yongjun Wang (Y)

National Clinical Research Center for Neurological Diseases, Beijing Tiantan Hospital, Capital Medical University, Beijing, China; Department of Neurology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China; Advanced Innovation Center for Human Brain Protection, Capital Medical University, Beijing, China. Electronic address: yongjunwang@ncrcnd.org.cn.

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