Dyskerin Downregulation Can Induce ER Stress and Promote Autophagy via AKT-mTOR Signaling Deregulation.

DKC1 LC3 puncta UPR autophagic flux dyskeratosis congenita

Journal

Biomedicines
ISSN: 2227-9059
Titre abrégé: Biomedicines
Pays: Switzerland
ID NLM: 101691304

Informations de publication

Date de publication:
08 May 2022
Historique:
received: 31 03 2022
revised: 28 04 2022
accepted: 04 05 2022
entrez: 28 5 2022
pubmed: 29 5 2022
medline: 29 5 2022
Statut: epublish

Résumé

Dyskerin is an evolutionarily conserved nucleolar protein implicated in a wide range of fundamental biological roles, including telomere maintenance and ribosome biogenesis. Germline mutations of

Identifiants

pubmed: 35625829
pii: biomedicines10051092
doi: 10.3390/biomedicines10051092
pmc: PMC9138296
pii:
doi:

Types de publication

Journal Article

Langues

eng

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Auteurs

Daniela Maiello (D)

Department of Biology, University of Naples Federico II, 80126 Naples, Italy.

Marianna Varone (M)

Department of Biology, University of Naples Federico II, 80126 Naples, Italy.

Rosario Vicidomini (R)

Section on Cellular Communication, Eunice Kennedy Shriver National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD 20892, USA.

Valentina Belli (V)

Medical Oncology, Department of Precision Medicine, Università degli Studi della Campania "Luigi Vanvitelli", 80131 Naples, Italy.

Marina De Rosa (M)

Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, 80131 Naples, Italy.

Paola Dama (P)

School of Life Sciences, University of Sussex, Brighton BN1 9QG, UK.

Maria Furia (M)

Department of Biology, University of Naples Federico II, 80126 Naples, Italy.

Mimmo Turano (M)

Department of Biology, University of Naples Federico II, 80126 Naples, Italy.

Classifications MeSH