SARS-COV-2 vaccine responses in renal patient populations.


Journal

BMC nephrology
ISSN: 1471-2369
Titre abrégé: BMC Nephrol
Pays: England
ID NLM: 100967793

Informations de publication

Date de publication:
31 05 2022
Historique:
received: 24 12 2021
accepted: 11 04 2022
entrez: 1 6 2022
pubmed: 2 6 2022
medline: 3 6 2022
Statut: epublish

Résumé

Dialysis patients and immunosuppressed renal patients are at increased risk of COVID-19 and were excluded from vaccine trials. We conducted a prospective multicentre study to assess SARS-CoV-2 vaccine antibody responses in dialysis patients and renal transplant recipients, and patients receiving immunosuppression for autoimmune disease. Patients were recruited from three UK centres (ethics:20/EM/0180) and compared to healthy controls (ethics:17/EE/0025). SARS-CoV-2 IgG antibodies to spike protein were measured using a multiplex Luminex assay, after first and second doses of Pfizer BioNTech BNT162b2(Pfizer) or Oxford-AstraZeneca ChAdOx1nCoV-19(AZ) vaccine. Six hundred ninety-two patients were included (260 dialysis, 209 transplant, 223 autoimmune disease (prior rituximab 128(57%)) and 144 healthy controls. 299(43%) patients received Pfizer vaccine and 379(55%) received AZ. Following two vaccine doses, positive responses occurred in 96% dialysis, 52% transplant, 70% autoimmune patients and 100% of healthy controls. In dialysis patients, higher antibody responses were observed with the Pfizer vaccination. Predictors of poor antibody response were triple immunosuppression (adjusted odds ratio [aOR]0.016;95%CI0.002-0.13;p < 0.001) and mycophenolate mofetil (MMF) (aOR0.2;95%CI 0.1-0.42;p < 0.001) in transplant patients; rituximab within 12 months in autoimmune patients (aOR0.29;95%CI 0.008-0.096;p < 0.001) and patients receiving immunosuppression with eGFR 15-29 ml/min (aOR0.031;95%CI 0.11-0.84;p = 0.021). Lower antibody responses were associated with a higher chance of a breakthrough infection. Amongst dialysis, kidney transplant and autoimmune populations SARS-CoV-2 vaccine antibody responses are reduced compared to healthy controls. A reduced response to vaccination was associated with rituximab, MMF, triple immunosuppression CKD stage 4. Vaccine responses increased after the second dose, suggesting low-responder groups should be prioritised for repeated vaccination. Greater antibody responses were observed with the mRNA Pfizer vaccine compared to adenovirus AZ vaccine in dialysis patients suggesting that Pfizer SARS-CoV-2 vaccine should be the preferred vaccine choice in this sub-group.

Sections du résumé

BACKGROUND
Dialysis patients and immunosuppressed renal patients are at increased risk of COVID-19 and were excluded from vaccine trials. We conducted a prospective multicentre study to assess SARS-CoV-2 vaccine antibody responses in dialysis patients and renal transplant recipients, and patients receiving immunosuppression for autoimmune disease.
METHODS
Patients were recruited from three UK centres (ethics:20/EM/0180) and compared to healthy controls (ethics:17/EE/0025). SARS-CoV-2 IgG antibodies to spike protein were measured using a multiplex Luminex assay, after first and second doses of Pfizer BioNTech BNT162b2(Pfizer) or Oxford-AstraZeneca ChAdOx1nCoV-19(AZ) vaccine.
RESULTS
Six hundred ninety-two patients were included (260 dialysis, 209 transplant, 223 autoimmune disease (prior rituximab 128(57%)) and 144 healthy controls. 299(43%) patients received Pfizer vaccine and 379(55%) received AZ. Following two vaccine doses, positive responses occurred in 96% dialysis, 52% transplant, 70% autoimmune patients and 100% of healthy controls. In dialysis patients, higher antibody responses were observed with the Pfizer vaccination. Predictors of poor antibody response were triple immunosuppression (adjusted odds ratio [aOR]0.016;95%CI0.002-0.13;p < 0.001) and mycophenolate mofetil (MMF) (aOR0.2;95%CI 0.1-0.42;p < 0.001) in transplant patients; rituximab within 12 months in autoimmune patients (aOR0.29;95%CI 0.008-0.096;p < 0.001) and patients receiving immunosuppression with eGFR 15-29 ml/min (aOR0.031;95%CI 0.11-0.84;p = 0.021). Lower antibody responses were associated with a higher chance of a breakthrough infection.
CONCLUSIONS
Amongst dialysis, kidney transplant and autoimmune populations SARS-CoV-2 vaccine antibody responses are reduced compared to healthy controls. A reduced response to vaccination was associated with rituximab, MMF, triple immunosuppression CKD stage 4. Vaccine responses increased after the second dose, suggesting low-responder groups should be prioritised for repeated vaccination. Greater antibody responses were observed with the mRNA Pfizer vaccine compared to adenovirus AZ vaccine in dialysis patients suggesting that Pfizer SARS-CoV-2 vaccine should be the preferred vaccine choice in this sub-group.

Identifiants

pubmed: 35641961
doi: 10.1186/s12882-022-02792-w
pii: 10.1186/s12882-022-02792-w
pmc: PMC9153874
doi:

Substances chimiques

COVID-19 Vaccines 0
Viral Vaccines 0
Rituximab 4F4X42SYQ6
Mycophenolic Acid HU9DX48N0T
BNT162 Vaccine N38TVC63NU

Types de publication

Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

199

Informations de copyright

© 2022. The Author(s).

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Auteurs

Rona M Smith (RM)

Department of Medicine, University of Cambridge, Cambridge, UK.
Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.
Department of Renal Medicine, Addenbrooke's Hospital, Hills Road, Box 118, Cambridge, CB2 0QQ, UK.

Daniel J Cooper (DJ)

Department of Medicine, University of Cambridge, Cambridge, UK.
Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Rainer Doffinger (R)

Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Hannah Stacey (H)

Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Abdulrahman Al-Mohammad (A)

Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Ian Goodfellow (I)

Division of Virology, Department of Pathology, University of Cambridge, Cambridge, UK.

Stephen Baker (S)

Department of Medicine, University of Cambridge, Cambridge, UK.

Sara Lear (S)

Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Myra Hosmilo (M)

Division of Virology, Department of Pathology, University of Cambridge, Cambridge, UK.

Nicholas Pritchard (N)

Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Nicholas Torpey (N)

Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

David Jayne (D)

Department of Medicine, University of Cambridge, Cambridge, UK.
Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Vivien Yiu (V)

West Suffolk Hospital NHS Foundation Trust, Bury St Edmunds, UK.

Anil Chalisey (A)

Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Jacinta Lee (J)

Department of Medicine, University of Cambridge, Cambridge, UK.

Enric Vilnar (E)

East and North Hertfordshire NHS Trust, Stevenage, UK.

Chee Kay Cheung (CK)

Department of Cardiovascular Sciences, University of Leicester, Leicester, UK.
University Hospitals of Leicester NHS Trust, Leicester, UK.

Rachel B Jones (RB)

Department of Medicine, University of Cambridge, Cambridge, UK.
Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

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