Comprehensive Transcriptional Profiling and Mouse Phenotyping Reveals Dispensable Role for Adipose Tissue Selective Long Noncoding RNA

adipose tissue remodelling brown adipose tissue long noncoding RNAs

Journal

Non-coding RNA
ISSN: 2311-553X
Titre abrégé: Noncoding RNA
Pays: Switzerland
ID NLM: 101652294

Informations de publication

Date de publication:
06 May 2022
Historique:
received: 11 04 2022
revised: 29 04 2022
accepted: 04 05 2022
entrez: 1 6 2022
pubmed: 2 6 2022
medline: 2 6 2022
Statut: epublish

Résumé

Cold and nutrient-activated brown adipose tissue (BAT) is capable of increasing systemic energy expenditure via the uncoupled respiration and secretion of endocrine factors, thereby protecting mice against diet-induced obesity and improving insulin response and glucose tolerance in men. Long non-coding RNAs (lncRNAs) have recently been identified as fine-tuning regulators of cellular function. While certain lncRNAs have been functionally characterised in adipose tissue, their overall contribution in the activation of BAT remains elusive. We identified lncRNAs correlating to interscapular brown adipose tissue (iBAT) function in a high fat diet (HFD) and cold stressed mice. We focused on

Identifiants

pubmed: 35645339
pii: ncrna8030032
doi: 10.3390/ncrna8030032
pmc: PMC9149892
pii:
doi:

Types de publication

Journal Article

Langues

eng

Subventions

Organisme : European Research Council
ID : 675014
Pays : International
Organisme : Danish Diabetes Academy
ID : 00000
Organisme : Novo Nordisk Foundation
ID : NNF18OC0033444

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Auteurs

Christoph Andreas Engelhard (CA)

Department for Biochemistry and Molecular Biology (BMB), University of Southern Denmark, Campusvej 55, 5230 Odense, Denmark.

Chien Huang (C)

Department for Biochemistry and Molecular Biology (BMB), University of Southern Denmark, Campusvej 55, 5230 Odense, Denmark.
Laboratory of Animal Physiology, Department of Animal Science and Technology, National Taiwan University, Taipei 10617, Taiwan.

Sajjad Khani (S)

Max Planck Institute for Metabolism Research, Gleueler Strasse 50, 50931 Köln, Germany.
Institute for Diabetes and Cancer (IDC), Helmholtz Zentrum München, German Research Center for Environmental Health, 85764 Neuherberg, Germany.
Cluster of Excellence Cellular Stress Responses in Aging-Associated Diseases (CECAD), Faculty of Medicine, University Hospital of Cologne, University of Cologne, Joseph-Stelzmann-Str. 26, 50931 Cologne, Germany.

Petr Kasparek (P)

Czech Centre for Phenogenomics, Institute of Molecular Genetics of the Czech Academy of Sciences, Prumyslova 595, 25250 Vestec, Czech Republic.

Jan Prochazka (J)

Czech Centre for Phenogenomics, Institute of Molecular Genetics of the Czech Academy of Sciences, Prumyslova 595, 25250 Vestec, Czech Republic.

Jan Rozman (J)

Czech Centre for Phenogenomics, Institute of Molecular Genetics of the Czech Academy of Sciences, Prumyslova 595, 25250 Vestec, Czech Republic.

David Pajuelo Reguera (DP)

Czech Centre for Phenogenomics, Institute of Molecular Genetics of the Czech Academy of Sciences, Prumyslova 595, 25250 Vestec, Czech Republic.

Radislav Sedlacek (R)

Czech Centre for Phenogenomics, Institute of Molecular Genetics of the Czech Academy of Sciences, Prumyslova 595, 25250 Vestec, Czech Republic.

Jan-Wilhelm Kornfeld (JW)

Department for Biochemistry and Molecular Biology (BMB), University of Southern Denmark, Campusvej 55, 5230 Odense, Denmark.
Max Planck Institute for Metabolism Research, Gleueler Strasse 50, 50931 Köln, Germany.

Classifications MeSH