The frontostriatal subtype of mild cognitive impairment in Parkinson's disease, but not the posterior cortical one, is associated with specific EEG alterations.

Cognition Dual syndrome hypothesis Functional connectivity PD-MCI Spectral decomposition

Journal

Cortex; a journal devoted to the study of the nervous system and behavior
ISSN: 1973-8102
Titre abrégé: Cortex
Pays: Italy
ID NLM: 0100725

Informations de publication

Date de publication:
08 2022
Historique:
received: 21 10 2021
revised: 27 01 2022
accepted: 07 04 2022
pubmed: 7 6 2022
medline: 29 6 2022
entrez: 6 6 2022
Statut: ppublish

Résumé

The 'dual syndrome' hypothesis states that two cognitive subtypes can be distinguished in mild cognitive impairment in Parkinson's disease (PD-MCI): a frontostriatal one, characterized by attentional and/or executive deficits, and a posterior cortical one, characterized by visuospatial, memory and/or language deficits. The latter type has been associated with a higher risk of earlier development of PD dementia. The functional bases of these subtypes remain partly unknown. To identify EEG modifications associated with PD-MCI subtypes. 75 non-demented PD patients underwent a comprehensive neuropsychological assessment and a high-density EEG. They were classified as having normal cognition (PD-NC; n = 37), PD-MCI with a frontostriatal subtype (PD-FS; n = 11) or PD-MCI with a posterior cortical subtype (PD-PC; n = 27). Two EEG analyses were performed: (a) spectral powers quantification and (b) functional connectivity analysis. PD-FS patients displayed spectral and functional EEG alterations, namely (a) higher powers in the theta and delta bands, (b) lower powers in the beta2 band and (c) lower functional connectivity in the beta2 band compared to PD-NC and PD-PC patients. These alterations were mainly located in the frontal, limbic and parietal regions. There were no significant differences between PD-NC and PD-PC. EEG alterations previously reported in PD-MCI may only concern the frontostriatal subtype, and not the posterior-cortical subtype. This provides evidence for the dual syndrome hypothesis and emphasizes the importance of identifying PD-MCI subtypes. It also shows the promising potential of EEG to discriminate between PD-MCI subtypes.

Sections du résumé

BACKGROUND
The 'dual syndrome' hypothesis states that two cognitive subtypes can be distinguished in mild cognitive impairment in Parkinson's disease (PD-MCI): a frontostriatal one, characterized by attentional and/or executive deficits, and a posterior cortical one, characterized by visuospatial, memory and/or language deficits. The latter type has been associated with a higher risk of earlier development of PD dementia. The functional bases of these subtypes remain partly unknown.
OBJECTIVE
To identify EEG modifications associated with PD-MCI subtypes.
METHODS
75 non-demented PD patients underwent a comprehensive neuropsychological assessment and a high-density EEG. They were classified as having normal cognition (PD-NC; n = 37), PD-MCI with a frontostriatal subtype (PD-FS; n = 11) or PD-MCI with a posterior cortical subtype (PD-PC; n = 27). Two EEG analyses were performed: (a) spectral powers quantification and (b) functional connectivity analysis.
RESULTS
PD-FS patients displayed spectral and functional EEG alterations, namely (a) higher powers in the theta and delta bands, (b) lower powers in the beta2 band and (c) lower functional connectivity in the beta2 band compared to PD-NC and PD-PC patients. These alterations were mainly located in the frontal, limbic and parietal regions. There were no significant differences between PD-NC and PD-PC.
CONCLUSION
EEG alterations previously reported in PD-MCI may only concern the frontostriatal subtype, and not the posterior-cortical subtype. This provides evidence for the dual syndrome hypothesis and emphasizes the importance of identifying PD-MCI subtypes. It also shows the promising potential of EEG to discriminate between PD-MCI subtypes.

Identifiants

pubmed: 35667287
pii: S0010-9452(22)00129-0
doi: 10.1016/j.cortex.2022.04.015
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

166-177

Informations de copyright

Copyright © 2022 Elsevier Ltd. All rights reserved.

Auteurs

Nacim Betrouni (N)

Univ. Lille, U1172 - LilNCog - Lille Neuroscience & Cognition, F-59000, Lille, France; Inserm, U1172, F-59000, Lille, France. Electronic address: nacim.betrouni@inserm.fr.

Quentin Devignes (Q)

Univ. Lille, U1172 - LilNCog - Lille Neuroscience & Cognition, F-59000, Lille, France; Inserm, U1172, F-59000, Lille, France.

Madli Bayot (M)

Univ. Lille, U1172 - LilNCog - Lille Neuroscience & Cognition, F-59000, Lille, France; Inserm, U1172, F-59000, Lille, France; CHU Lille, Clinical Neurophysiology Department, F-59000, Lille, France.

Philippe Derambure (P)

Univ. Lille, U1172 - LilNCog - Lille Neuroscience & Cognition, F-59000, Lille, France; Inserm, U1172, F-59000, Lille, France; CHU Lille, Clinical Neurophysiology Department, F-59000, Lille, France.

Luc Defebvre (L)

Univ. Lille, U1172 - LilNCog - Lille Neuroscience & Cognition, F-59000, Lille, France; Inserm, U1172, F-59000, Lille, France; CHU Lille, Neurology and Movement Disorders Department, F-59000, Lille, France.

Albert Fg Leentjens (AF)

Maastricht University Medical Center, Maastricht, the Netherlands.

Arnaud Delval (A)

Univ. Lille, U1172 - LilNCog - Lille Neuroscience & Cognition, F-59000, Lille, France; Inserm, U1172, F-59000, Lille, France; CHU Lille, Clinical Neurophysiology Department, F-59000, Lille, France.

Kathy Dujardin (K)

Univ. Lille, U1172 - LilNCog - Lille Neuroscience & Cognition, F-59000, Lille, France; Inserm, U1172, F-59000, Lille, France; CHU Lille, Neurology and Movement Disorders Department, F-59000, Lille, France.

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