Heart rate changes associated with autonomic dysreflexia in daily life of individuals with chronic spinal cord injury.


Journal

Spinal cord
ISSN: 1476-5624
Titre abrégé: Spinal Cord
Pays: England
ID NLM: 9609749

Informations de publication

Date de publication:
11 2022
Historique:
received: 09 10 2021
accepted: 30 05 2022
revised: 29 05 2022
pubmed: 11 6 2022
medline: 8 11 2022
entrez: 10 6 2022
Statut: ppublish

Résumé

Secondary data analysis. To characterize heart rate (HR) changes during autonomic dysreflexia (AD) in daily life for individuals with chronic spinal cord injury (SCI). University-based laboratory/community-based outpatient. Cardiovascular data, previously collected during a 24-h ambulatory surveillance period in individuals with chronic SCI, were assessed. Any systolic blood pressure (SBP) increase ≥20 mmHg from baseline was identified and categorized into confirmed AD (i.e., diarized trigger), unknown (i.e., no diary entry), or unlikely AD (i.e., potential exertion driven SBP increase) groups. SBP-associated HR changes were categorized as unchanged, increased or decreased compared to baseline. Forty-five individuals [8 females, median age and time since injury of 43 years (lower and upper quartiles 36-50) and 17 years (6-23), respectively], were included for analysis. Overall, 797 episodes of SBP increase above AD threshold were identified and classified as confirmed (n = 250, 31.4%), unknown (n = 472, 59.2%) or unlikely (n = 75, 9.4%). The median number of episodes per individual within the 24-h period was 13 (8-28). HR-decrease/increase ratio was 3:1 for confirmed and unknown, and 1.5:1 for unlikely episodes. HR changes resulting in brady-/tachycardia were 34.4%/2.8% for confirmed, 39.6%/3.4% unknown, and 26.7%/9.3% for unlikely episodes, respectively. Our findings suggest that the majority of confirmed AD episodes are associated with a HR decrease. Using wearable-sensors-derived measures of physical activity in future studies could provide a more detailed characterization of HR changes during AD and improve AD identification.

Identifiants

pubmed: 35680988
doi: 10.1038/s41393-022-00820-y
pii: 10.1038/s41393-022-00820-y
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1030-1036

Informations de copyright

© 2022. The Author(s), under exclusive licence to International Spinal Cord Society.

Références

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Auteurs

Belinda Yee (B)

International Collaboration on Repair Discoveries (ICORD), Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada.
MD Undergraduate Program, Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada.

Tom E Nightingale (TE)

International Collaboration on Repair Discoveries (ICORD), Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada.
School of Sport, Exercise and Rehabilitation Sciences, University of Birmingham, Birmingham, United Kingdom.
Centre for Trauma Sciences Research, University of Birmingham, Edgbaston, Birmingham, United Kingdom.

Andrea L Ramirez (AL)

International Collaboration on Repair Discoveries (ICORD), Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada.

Matthias Walter (M)

International Collaboration on Repair Discoveries (ICORD), Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada. dr.matze@gmx.de.
Department of Urology, University Hospital Basel, University of Basel, Basel, Switzerland. dr.matze@gmx.de.

Andrei V Krassioukov (AV)

International Collaboration on Repair Discoveries (ICORD), Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada. krassioukov@icord.org.
G.F. Strong Rehabilitation Centre, Vancouver, British Columbia, Canada. krassioukov@icord.org.
Division of Physical Medicine and Rehabilitation, Faculty of Medicine, University of British Columbia, Vancouver, British Columbia, Canada. krassioukov@icord.org.

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