Alzheimer's Disease with Epileptiform EEG Activity: Abnormal Cortical Sources of Resting State Delta Rhythms in Patients with Amnesic Mild Cognitive Impairment.

Epileptiform EEG activity exact low-resolution brain electromagnetic source tomography mild cognitive impairment due to Alzheimer’s disease resting state electroencephalographic rhythms

Journal

Journal of Alzheimer's disease : JAD
ISSN: 1875-8908
Titre abrégé: J Alzheimers Dis
Pays: Netherlands
ID NLM: 9814863

Informations de publication

Date de publication:
2022
Historique:
pubmed: 14 6 2022
medline: 10 8 2022
entrez: 13 6 2022
Statut: ppublish

Résumé

Patients with amnesic mild cognitive impairment due to Alzheimer's disease (ADMCI) typically show a "slowing" of cortical resting-state eyes-closed electroencephalographic (rsEEG) rhythms. Some of them also show subclinical, non-convulsive, and epileptiform EEG activity (EEA) with an unclear relationship with that "slowing." Here we tested the hypothesis that the "slowing" of rsEEG rhythms is related to EEA in ADMCI patients. Clinical and instrumental datasets in 62 ADMCI patients and 38 normal elderly (Nold) subjects were available in a national archive. No participant had received a clinical diagnosis of epilepsy. The eLORETA freeware estimated rsEEG cortical sources. The area under the receiver operating characteristic curve (AUROCC) indexed the accuracy of eLORETA solutions in the classification between ADMCI-EEA and ADMCI-noEEA individuals. EEA was observed in 15% (N = 8) of the ADMCI patients. The ADMCI-EEA group showed: 1) more abnormal Aβ42 levels in the cerebrospinal fluid as compared to the ADMCI-noEEA group and 2) higher temporal and occipital delta (<4 Hz) rsEEG source activities as compared to the ADMCI-noEEA and Nold groups. Those source activities showed moderate accuracy (AUROCC = 0.70-0.75) in the discrimination between ADMCI-noEEA versus ADMCI-EEA individuals. It can be speculated that in ADMCI-EEA patients, AD-related amyloid neuropathology may be related to an over-excitation in neurophysiological low-frequency (delta) oscillatory mechanisms underpinning cortical arousal and quiet vigilance.

Sections du résumé

BACKGROUND
Patients with amnesic mild cognitive impairment due to Alzheimer's disease (ADMCI) typically show a "slowing" of cortical resting-state eyes-closed electroencephalographic (rsEEG) rhythms. Some of them also show subclinical, non-convulsive, and epileptiform EEG activity (EEA) with an unclear relationship with that "slowing."
OBJECTIVE
Here we tested the hypothesis that the "slowing" of rsEEG rhythms is related to EEA in ADMCI patients.
METHODS
Clinical and instrumental datasets in 62 ADMCI patients and 38 normal elderly (Nold) subjects were available in a national archive. No participant had received a clinical diagnosis of epilepsy. The eLORETA freeware estimated rsEEG cortical sources. The area under the receiver operating characteristic curve (AUROCC) indexed the accuracy of eLORETA solutions in the classification between ADMCI-EEA and ADMCI-noEEA individuals.
RESULTS
EEA was observed in 15% (N = 8) of the ADMCI patients. The ADMCI-EEA group showed: 1) more abnormal Aβ42 levels in the cerebrospinal fluid as compared to the ADMCI-noEEA group and 2) higher temporal and occipital delta (<4 Hz) rsEEG source activities as compared to the ADMCI-noEEA and Nold groups. Those source activities showed moderate accuracy (AUROCC = 0.70-0.75) in the discrimination between ADMCI-noEEA versus ADMCI-EEA individuals.
CONCLUSION
It can be speculated that in ADMCI-EEA patients, AD-related amyloid neuropathology may be related to an over-excitation in neurophysiological low-frequency (delta) oscillatory mechanisms underpinning cortical arousal and quiet vigilance.

Identifiants

pubmed: 35694930
pii: JAD220442
doi: 10.3233/JAD-220442
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

903-931

Auteurs

Claudio Babiloni (C)

Department of Physiology and Pharmacology "Vittorio Erspamer", Sapienza University of Rome, Rome, Italy.
Hospital San Raffaele Cassino, Cassino (FR), Italy.

Giuseppe Noce (G)

IRCCS Synlab SDN, Naples, Italy.

Carlo Di Bonaventura (C)

Epilepsy Unit, Department of Neurosciences/Mental Health, Sapienza University of Rome, Rome, Italy.

Roberta Lizio (R)

IRCCS Synlab SDN, Naples, Italy.

Ali Eldellaa (A)

Department of Physiology and Pharmacology "Vittorio Erspamer", Sapienza University of Rome, Rome, Italy.

Federico Tucci (F)

Department of Physiology and Pharmacology "Vittorio Erspamer", Sapienza University of Rome, Rome, Italy.

Enrico M Salamone (EM)

Department of Physiology and Pharmacology "Vittorio Erspamer", Sapienza University of Rome, Rome, Italy.
Epilepsy Unit, Department of Neurosciences/Mental Health, Sapienza University of Rome, Rome, Italy.

Raffaele Ferri (R)

Oasi Research Institute - IRCCS, Troina, Italy.

Andrea Soricelli (A)

IRCCS Synlab SDN, Naples, Italy.
Department of Motor Sciences and Healthiness, University of Naples Parthenope, Naples, Italy.

Flavio Nobili (F)

Clinical Neurology, IRCCS Hospital Policlinico San Martino, Genoa, Italy.
Department of Neuroscience (DiNOGMI), University of Genoa, Genoa, Italy.

Francesco Famà (F)

Clinical Neurology, IRCCS Hospital Policlinico San Martino, Genoa, Italy.

Dario Arnaldi (D)

Clinical Neurology, IRCCS Hospital Policlinico San Martino, Genoa, Italy.

Eleonora Palma (E)

Department of Physiology and Pharmacology "Vittorio Erspamer", Sapienza University of Rome, Rome, Italy.
Pasteur Institute-Cenci Bolognetti Foundation, Rome, Italy.

Pierangelo Cifelli (P)

IRCCS Neuromed, Pozzilli, (IS), Italy.
Department of Applied and Biotechnological Clinical Sciences, University of L'Aquila, L'Aquila, Italy.

Moira Marizzoni (M)

Laboratory of Alzheimer's Neuroimaging and Epidemiology, IRCCS Istituto Centro San Giovanni di Dio Fatebenefratelli, Brescia, Italy.

Fabrizio Stocchi (F)

IRCCS San Raffaele Roma, Rome, Italy.

Giuseppe Bruno (G)

Department of Human Neurosciences, Sapienza University of Rome, Rome, Italy.

Giancarlo Di Gennaro (G)

IRCCS Neuromed, Pozzilli, (IS), Italy.

Giovanni B Frisoni (GB)

Laboratory of Alzheimer's Neuroimaging and Epidemiology, IRCCS Istituto Centro San Giovanni di Dio Fatebenefratelli, Brescia, Italy.
Memory Clinic and LANVIE - Laboratory of Neuroimaging of Aging, University Hospitals and University of Geneva, Geneva, Switzerland.

Claudio Del Percio (C)

Department of Physiology and Pharmacology "Vittorio Erspamer", Sapienza University of Rome, Rome, Italy.

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