Reconstruction of the origin and dispersal of the worldwide dominant Hepatitis B Virus subgenotype D1.

Bayesian inference D1 HBV MCMC full genome phylodynamics temporal signal

Journal

Virus evolution
ISSN: 2057-1577
Titre abrégé: Virus Evol
Pays: England
ID NLM: 101664675

Informations de publication

Date de publication:
2022
Historique:
received: 27 09 2021
revised: 18 02 2022
accepted: 07 04 2022
entrez: 17 6 2022
pubmed: 18 6 2022
medline: 18 6 2022
Statut: epublish

Résumé

Hepatitis B is a potentially life-threatening liver infection caused by the hepatitis B virus (HBV). HBV-D1 is the dominant subgenotype in the Mediterranean basin, Eastern Europe, and Asia. However, little is currently known about its evolutionary history and spatio-temporal dynamics. We use Bayesian phylodynamic inference to investigate the temporal history of HBV-D1, for which we calibrate the molecular clock using ancient sequences, and reconstruct the viral global spatial dynamics based, for the first time, on full-length publicly available HBV-D1 genomes from a wide range of sampling dates. We pinpoint the origin of HBV subgenotype D1 before the current era (BCE) in Turkey/Anatolia. The spatial reconstructions reveal global viral transmission with a high degree of mixing. By combining modern-day and ancient sequences, we ensure sufficient temporal signal in HBV-D1 data to enable Bayesian phylodynamic inference using a molecular clock for time calibration. Our results shed light on the worldwide HBV-D1 epidemics and suggest that this originally Middle Eastern virus significantly affects more distant countries, such as those in mainland Europe.

Identifiants

pubmed: 35712523
doi: 10.1093/ve/veac028
pii: veac028
pmc: PMC9194798
doi:

Types de publication

Journal Article Review

Langues

eng

Pagination

veac028

Commentaires et corrections

Type : ErratumIn

Informations de copyright

© The Author(s) 2022. Published by Oxford University Press.

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Auteurs

Nídia Sequeira Trovão (NS)

Department of Microbiology, Immunology and Transplantation, KU Leuven, Rega Institute, Laboratory for Clinical and Epidemiological Virology, Herestraat 49, Leuven BE-3000, Belgium.

Marijn Thijssen (M)

Department of Microbiology, Immunology and Transplantation, KU Leuven, Rega Institute, Laboratory for Clinical and Epidemiological Virology, Herestraat 49, Leuven BE-3000, Belgium.

Bram Vrancken (B)

Department of Microbiology, Immunology and Transplantation, KU Leuven, Rega Institute, Laboratory for Clinical and Epidemiological Virology, Herestraat 49, Leuven BE-3000, Belgium.

Andrea-Clemencia Pineda-Peña (AC)

Global Health and Tropical Medicine, GHTM, Instituto de Higiene e Medicina Tropical, IHMT; Universidade Nova de Lisboa, UNL, Portugal Rua da Junqueira No 100, Lisbon 1349-008, Portugal.

Thomas Mina (T)

Nonis Lab Microbiology - Virology Unit, Gregori Afxentiou 5, Limassol 4003, Cyprus.

Samad Amini-Bavil-Olyaee (S)

Cellular Sciences Department, Biosafety Development Group, Amgen Inc., One Amgen Center Drive, Thousand Oaks, CA 91320, USA.

Philippe Lemey (P)

Department of Microbiology, Immunology and Transplantation, KU Leuven, Rega Institute, Laboratory for Clinical and Epidemiological Virology, Herestraat 49, Leuven BE-3000, Belgium.

Guy Baele (G)

Department of Microbiology, Immunology and Transplantation, KU Leuven, Rega Institute, Laboratory for Clinical and Epidemiological Virology, Herestraat 49, Leuven BE-3000, Belgium.

Mahmoud Reza Pourkarim (MR)

Department of Microbiology, Immunology and Transplantation, KU Leuven, Rega Institute, Laboratory for Clinical and Epidemiological Virology, Herestraat 49, Leuven BE-3000, Belgium.

Classifications MeSH