Phenotyping Young GluA1 Deficient Mice - A Behavioral Characterization in a Genetic Loss-of-Function Model.
3R
GRIA1
genetic mouse model
glucocorticoids
glutamate
knockout
schizophrenia
severity assessment
Journal
Frontiers in behavioral neuroscience
ISSN: 1662-5153
Titre abrégé: Front Behav Neurosci
Pays: Switzerland
ID NLM: 101477952
Informations de publication
Date de publication:
2022
2022
Historique:
received:
16
02
2022
accepted:
29
04
2022
entrez:
20
6
2022
pubmed:
21
6
2022
medline:
21
6
2022
Statut:
epublish
Résumé
Alterations of glutamatergic neurotransmission have been implicated in neurodevelopmental and neuropsychiatric disorders. Mice lacking the GluA1 AMPA receptor subunit, encoded by the
Identifiants
pubmed: 35722188
doi: 10.3389/fnbeh.2022.877094
pmc: PMC9204703
doi:
Types de publication
Journal Article
Langues
eng
Pagination
877094Informations de copyright
Copyright © 2022 Reiber, Stirling, Sprengel, Gass, Palme and Potschka.
Déclaration de conflit d'intérêts
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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