Chemokines and chemokine receptors in allergic rhinitis: from mediators to potential therapeutic targets.


Journal

European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery
ISSN: 1434-4726
Titre abrégé: Eur Arch Otorhinolaryngol
Pays: Germany
ID NLM: 9002937

Informations de publication

Date de publication:
Nov 2022
Historique:
received: 17 05 2022
accepted: 30 05 2022
pubmed: 23 6 2022
medline: 1 10 2022
entrez: 22 6 2022
Statut: ppublish

Résumé

Allergic rhinitis (AR) is an immune-mediated inflammatory condition characterized by immune cell infiltration of the nasal mucosa, with symptoms of rhinorrhea, sneezing, nasal obstruction, and itchiness. Currently, common medication for AR is anti-inflammatory treatment including intranasal steroids, oral, or intranasal anti-histamines, and immunotherapy. These strategies are effective to the majority of patients with AR, but some patients under medication cannot achieve symptom relieve and suffer from bothersome side effects, indicating a demand for novel anti-inflammatory treatment as alternatives. Chemokines, a complex superfamily of small, secreted proteins, were initially recognized for their chemotactic effects on various immune cells. Chemokines constitute both physiological and inflammatory cell positioning systems and mediate cell localization to certain sites via interaction with their receptors, which are expressed on responding cells. Chemokines and their receptors participate in the sensitization, early phase response, and late phase response of AR by promoting inflammatory cell recruitment, differentiation, and allergic mediator release. In this review, we first systemically summarize chemokines and chemokine receptors that are important in AR pathophysiology and then discuss potential strategies targeting chemokines and their receptors for AR therapy.

Identifiants

pubmed: 35732904
doi: 10.1007/s00405-022-07485-6
pii: 10.1007/s00405-022-07485-6
doi:

Substances chimiques

Anti-Inflammatory Agents 0
Chemokines 0
Receptors, Chemokine 0
Steroids 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

5089-5095

Subventions

Organisme : National Natural Science Foundation of China
ID : 81902133

Informations de copyright

© 2022. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.

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Auteurs

Zhan Li (Z)

Department of Laboratory Medicine, China Medical University, Shenyang, China.

Sihua Yu (S)

Department of Laboratory Medicine, China Medical University, Shenyang, China.

Yongjun Jiang (Y)

Department of Laboratory Medicine, China Medical University, Shenyang, China.
Department of Clinical Immunology, The First Affiliated Hospital of China Medical University, China Medical University, Shenyang, China.

Yajing Fu (Y)

Department of Laboratory Medicine, China Medical University, Shenyang, China. fufu80s@sina.com.
Department of Clinical Immunology, The First Affiliated Hospital of China Medical University, China Medical University, Shenyang, China. fufu80s@sina.com.

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