Severe Acute Respiratory Syndrome Coronavirus 2 Is Detected in the Gastrointestinal Tract of Asymptomatic Endoscopy Patients but Is Unlikely to Pose a Significant Risk to Healthcare Personnel.

COVID-19, coronavirus disease 2019 Colonic Liquid E gene, envelope gene Endoscopy GI, gastrointestinal N gene, nucleocapsid gene NP, nucleocapsid protein PFU, plaque-forming units SARS-CoV-2 SARS-CoV-2, severe acute respiratory syndrome coronavirus 2 Transmission Risk qRT-PCR, quantitative reverse transcription polymerase chain reaction

Journal

Gastro hep advances
ISSN: 2772-5723
Titre abrégé: Gastro Hep Adv
Pays: Netherlands
ID NLM: 9918350485906676

Informations de publication

Date de publication:
2022
Historique:
received: 17 03 2022
accepted: 03 06 2022
pubmed: 30 6 2022
medline: 30 6 2022
entrez: 29 6 2022
Statut: ppublish

Résumé

Recent evidence suggests that the gut is an additional target for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. However, whether SARS-CoV-2 spreads via gastrointestinal secretions remains unclear. To determine the prevalence of gastrointestinal SARS-CoV-2 infection in asymptomatic subjects, we analyzed gastrointestinal biopsy and liquid samples from endoscopy patients for the presence of SARS-CoV-2. We enrolled 100 endoscopic patients without known SARS-CoV-2 infection (cohort A) and 12 patients with a previous COVID-19 diagnosis (cohort B) in a cohort study performed at a regional hospital. Gastrointestinal biopsies and fluids were screened for SARS-CoV-2 by polymerase chain reaction (PCR), immunohistochemistry, and virus isolation assay, and the stability of SARS-CoV-2 in gastrointestinal liquids in vitro was analyzed. SARS-CoV-2 ribonucleic acid was detected by PCR in the colonic tissue of 1/100 patients in cohort A. In cohort B, 3 colonic liquid samples tested positive for SARS-CoV-2 by PCR and viral nucleocapsid protein was detected in the epithelium of the respective biopsy samples. However, no infectious virions were recovered from any samples. In vitro exposure of SARS-CoV-2 to colonic liquid led to a 4-log-fold reduction of infectious SARS-CoV-2 within 1 hour ( Overall, the persistent detection of SARS-CoV-2 in endoscopy samples after resolution of COVID-19 points to the gut as a long-term reservoir for SARS-CoV-2. Since no infectious virions were recovered and SARS-CoV-2 was rapidly inactivated in the presence of colon liquids, it is unlikely that performing endoscopic procedures is associated with a significant infection risk due to undiagnosed asymptomatic or persistent gastrointestinal SARS-CoV-2 infections.

Sections du résumé

Background and Aims UNASSIGNED
Recent evidence suggests that the gut is an additional target for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. However, whether SARS-CoV-2 spreads via gastrointestinal secretions remains unclear. To determine the prevalence of gastrointestinal SARS-CoV-2 infection in asymptomatic subjects, we analyzed gastrointestinal biopsy and liquid samples from endoscopy patients for the presence of SARS-CoV-2.
Methods UNASSIGNED
We enrolled 100 endoscopic patients without known SARS-CoV-2 infection (cohort A) and 12 patients with a previous COVID-19 diagnosis (cohort B) in a cohort study performed at a regional hospital. Gastrointestinal biopsies and fluids were screened for SARS-CoV-2 by polymerase chain reaction (PCR), immunohistochemistry, and virus isolation assay, and the stability of SARS-CoV-2 in gastrointestinal liquids in vitro was analyzed.
Results UNASSIGNED
SARS-CoV-2 ribonucleic acid was detected by PCR in the colonic tissue of 1/100 patients in cohort A. In cohort B, 3 colonic liquid samples tested positive for SARS-CoV-2 by PCR and viral nucleocapsid protein was detected in the epithelium of the respective biopsy samples. However, no infectious virions were recovered from any samples. In vitro exposure of SARS-CoV-2 to colonic liquid led to a 4-log-fold reduction of infectious SARS-CoV-2 within 1 hour (
Conclusion UNASSIGNED
Overall, the persistent detection of SARS-CoV-2 in endoscopy samples after resolution of COVID-19 points to the gut as a long-term reservoir for SARS-CoV-2. Since no infectious virions were recovered and SARS-CoV-2 was rapidly inactivated in the presence of colon liquids, it is unlikely that performing endoscopic procedures is associated with a significant infection risk due to undiagnosed asymptomatic or persistent gastrointestinal SARS-CoV-2 infections.

Identifiants

pubmed: 35765598
doi: 10.1016/j.gastha.2022.06.002
pii: S2772-5723(22)00097-8
pmc: PMC9225937
doi:

Types de publication

Journal Article

Langues

eng

Pagination

844-852

Subventions

Organisme : NIGMS NIH HHS
ID : R35 GM134867
Pays : United States
Organisme : NIBIB NIH HHS
ID : U01 EB029242
Pays : United States

Informations de copyright

© 2022 Published by Elsevier Inc. on behalf of the AGA Institute.

Références

Gut. 1988 Aug;29(8):1035-41
pubmed: 3410329
Nat Commun. 2021 Nov 16;12(1):6610
pubmed: 34785679
Mucosal Immunol. 2021 Nov;14(6):1381-1392
pubmed: 34420043
Gut. 2022 Jan;71(1):226-229
pubmed: 34083386
Gastroenterol Hepatol. 2022 Jan 22;:
pubmed: 35077722
Am J Gastroenterol. 2021 Jan 1;116(1):202-205
pubmed: 33079747
Gastroenterology. 2021 Jun;160(7):2435-2450.e34
pubmed: 33676971
Nat Med. 2020 Jul;26(7):1077-1083
pubmed: 32405028
J Med Virol. 2021 Oct;93(10):5917-5923
pubmed: 34139026
Am J Gastroenterol. 2021 Feb 1;116(2):433-434
pubmed: 33038127
Viruses. 2021 Dec 23;14(1):
pubmed: 35062227
Gastroenterology. 2021 Nov;161(5):1702-1704.e3
pubmed: 34280387
Eur J Clin Microbiol Infect Dis. 2020 Jun;39(6):1059-1061
pubmed: 32342252
Science. 2020 Jul 3;369(6499):50-54
pubmed: 32358202
Cell Rep. 2021 Jun 1;35(9):109197
pubmed: 34043946
Gastroenterology. 2020 May;158(6):1831-1833.e3
pubmed: 32142773
Cell Rep. 2020 Jul 7;32(1):107863
pubmed: 32610043
Lancet Gastroenterol Hepatol. 2020 May;5(5):434-435
pubmed: 32199469
Sci Immunol. 2020 May 13;5(47):
pubmed: 32404436
J Med Virol. 2022 Apr;94(4):1255-1256
pubmed: 34850421
Pediatr Infect Dis J. 2020 Sep;39(9):e249-e256
pubmed: 32618932
Gastroenterology. 2020 Jul;159(1):81-95
pubmed: 32251668
Gut. 2020 Jun;69(6):997-1001
pubmed: 32241899
Gastroenterology. 2021 Sep;161(3):1011-1029.e11
pubmed: 34029569
Lancet Oncol. 2020 Aug;21(8):1023-1034
pubmed: 32702310
Mol Syst Biol. 2021 Apr;17(4):e10232
pubmed: 33904651
Commun Biol. 2021 Nov 30;4(1):1343
pubmed: 34848826
Cell Rep Med. 2020 Sep 22;1(6):100098
pubmed: 32904687
Front Public Health. 2020 Nov 23;8:590041
pubmed: 33330334
Clin Infect Dis. 2021 Aug 2;73(3):361-366
pubmed: 32638022
Gastroenterology. 2022 May 1;:
pubmed: 35508284

Auteurs

Michelle D Cherne (MD)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Andrew B Gentry (AB)

Department of Gastroenterology, Bozeman Health Deaconess Hospital, Bozeman, Montana.

Anna Nemudraia (A)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Artem Nemudryi (A)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Jodi F Hedges (JF)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Heather Walk (H)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Karlin Blackwell (K)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Deann T Snyder (DT)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Maria Jerome (M)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Wyatt Madden (W)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.
Rollins School of Public Heath, Emory University, Atlanta, Georgia.

Marziah Hashimi (M)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

T Andrew Sebrell (TA)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

David B King (DB)

Department of Clinical Research, Bozeman Health Deaconess Hospital, Bozeman, Montana.

Raina K Plowright (RK)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Mark A Jutila (MA)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Blake Wiedenheft (B)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Diane Bimczok (D)

Department of Microbiology and Cell Biology, Montana State University, Bozeman, Montana.

Classifications MeSH