Maternal High-Fat diet During Pregnancy and Lactation Disrupts NMDA Receptor Expression and Spatial Memory in the Offspring.


Journal

Molecular neurobiology
ISSN: 1559-1182
Titre abrégé: Mol Neurobiol
Pays: United States
ID NLM: 8900963

Informations de publication

Date de publication:
Sep 2022
Historique:
received: 10 09 2021
accepted: 30 05 2022
pubmed: 1 7 2022
medline: 25 8 2022
entrez: 30 6 2022
Statut: ppublish

Résumé

The problem of an unbalanced diet, overly rich in fats, affects a significant proportion of the population, including women of childbearing age. Negative metabolic and endocrine outcomes for offspring associated with maternal high-fat diet during pregnancy and/or lactation are well documented in the literature. In this paper, we present our findings on the little-studied effects of this diet on NMDA receptors and cognitive functions in offspring. The subject of the study was the rat offspring born from dams fed a high-fat diet before mating and throughout pregnancy and lactation. Using a novel object location test, spatial memory impairment was detected in adolescent offspring as well as in young adult female offspring. The recognition memory of the adolescent and young adult offspring remained unaltered. We also found multiple alterations in the expression of the NMDA receptor subunits, NMDA receptor-associated scaffolding proteins, and selected microRNAs that regulate the activity of the NMDA receptor in the medial prefrontal cortex and the hippocampus of the offspring. Sex-dependent changes in glutamate levels were identified in extracellular fluid obtained from the medial prefrontal cortex and the hippocampus of the offspring. The obtained results indicate that a maternal high-fat diet during pregnancy and lactation can induce in the offspring memory disturbances accompanied by alterations in NMDA receptor expression.

Identifiants

pubmed: 35773600
doi: 10.1007/s12035-022-02908-1
pii: 10.1007/s12035-022-02908-1
doi:

Substances chimiques

Receptors, N-Methyl-D-Aspartate 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

5695-5721

Subventions

Organisme : National Science Center, Poland
ID : 2015/19/D/NZ7/00082

Informations de copyright

© 2022. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.

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Auteurs

Jozef Mizera (J)

Department of Toxicology, Jagiellonian University Medical College, Medyczna 9, 30-688, Kraków, PL, Poland.

Grzegorz Kazek (G)

Department of Pharmacodynamics, Jagiellonian University Medical College, Medyczna 9, 30-688, Kraków, PL, Poland.

Bartosz Pomierny (B)

Department of Biochemical Toxicology, Jagiellonian University Medical College, Medyczna 9, 30-688, Kraków, PL, Poland.

Beata Bystrowska (B)

Department of Biochemical Toxicology, Jagiellonian University Medical College, Medyczna 9, 30-688, Kraków, PL, Poland.

Ewa Niedzielska-Andres (E)

Department of Toxicology, Jagiellonian University Medical College, Medyczna 9, 30-688, Kraków, PL, Poland. ewa.niedzielska@uj.edu.pl.

Lucyna Pomierny-Chamiolo (L)

Department of Toxicology, Jagiellonian University Medical College, Medyczna 9, 30-688, Kraków, PL, Poland. ewa.niedzielska@uj.edu.pl.

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