Animals
Female
Mice
Autophagy
/ genetics
Autophagy-Related Protein 5
/ metabolism
Autophagy-Related Proteins
/ metabolism
Caenorhabditis elegans
/ metabolism
Granulosa Cells
/ metabolism
Primary Ovarian Insufficiency
/ genetics
Transcription Factors
/ metabolism
Vesicular Transport Proteins
/ metabolism
WT1 Proteins
/ genetics
Autophagy
ectopic P-granules protein 5 homolog
granulosa cell differentiation
primary ovarian insufficiency
wilms’ tumor gene 1
Journal
Autophagy
ISSN: 1554-8635
Titre abrégé: Autophagy
Pays: United States
ID NLM: 101265188
Informations de publication
Date de publication:
02 2023
02 2023
Historique:
pubmed:
6
7
2022
medline:
20
1
2023
entrez:
5
7
2022
Statut:
ppublish
Résumé
Primary ovarian insufficiency (POI), also known as premature ovarian failure, is an ovarian defect in humans characterized by the premature depletion of ovarian follicles before the age of 40. However, the mechanisms underlying POI remain largely unknown. Here, we show that knockout of
Identifiants
pubmed: 35786405
doi: 10.1080/15548627.2022.2094671
pmc: PMC9851269
doi:
Substances chimiques
Autophagy-Related Protein 5
0
Autophagy-Related Proteins
0
EPG5 protein, human
0
Transcription Factors
0
Vesicular Transport Proteins
0
WT1 protein, mouse
0
WT1 Proteins
0
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
644-659Références
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