Nestin as a Prognostic Biomarker in High-grade Epithelial Ovarian Cancer Treated by Neoadjuvant Chemotherapy.


Journal

Anticancer research
ISSN: 1791-7530
Titre abrégé: Anticancer Res
Pays: Greece
ID NLM: 8102988

Informations de publication

Date de publication:
Jul 2022
Historique:
received: 14 03 2022
revised: 05 05 2022
accepted: 01 06 2022
entrez: 5 7 2022
pubmed: 6 7 2022
medline: 7 7 2022
Statut: ppublish

Résumé

High-grade epithelial ovarian cancer (HGEOC) is a heterogeneous disease and among the deadliest types of cancer. It often acquires resistance to conventional chemotherapy and its prognosis remains highly poor. The tissue protein nestin, implicated in the assembly and disassembly of intermediate filaments, has been reported to be an unfavourable prognostic factor in several cancer types. We hypothesized that HGEOC progression is regulated by the proliferation of chemoresistant cancer stem cells, in which nestin might be implicated. This preliminary study aimed to evaluate nestin as a prognostic biomarker in HGEOC treated by neoadjuvant chemotherapy (NACT) followed by cytoreductive surgery. A retrospective study (2009-2019) was conducted on 92 patients with primary ovarian, fallopian tube or peritoneal HGEOC who underwent NACT followed by cytoreductive surgery. Nestin expression in tissue samples was semi-quantitatively evaluated defining nestin positivity for those with histochemical score ≥30. We then evaluated the prognostic value of nestin expression. The median progression-free survival was similar between nestin-positive (22 months) and nestin-negative (19 months) groups (p=0.57). Interestingly, the median overall survival was shorter for the nestin-positive group (48 vs. 67 months, respectively), however the difference did not reach statistical significance (p=0.43). Tissue nestin expression does not appear to be a relevant prognostic biomarker in HGEOC treated by NACT. However, we believe that prospective studies in larger cohorts should be conducted and evaluation of nestin in pre-NACT HGEOC samples needs to be explored.

Sections du résumé

BACKGROUND BACKGROUND
High-grade epithelial ovarian cancer (HGEOC) is a heterogeneous disease and among the deadliest types of cancer. It often acquires resistance to conventional chemotherapy and its prognosis remains highly poor. The tissue protein nestin, implicated in the assembly and disassembly of intermediate filaments, has been reported to be an unfavourable prognostic factor in several cancer types. We hypothesized that HGEOC progression is regulated by the proliferation of chemoresistant cancer stem cells, in which nestin might be implicated. This preliminary study aimed to evaluate nestin as a prognostic biomarker in HGEOC treated by neoadjuvant chemotherapy (NACT) followed by cytoreductive surgery.
PATIENTS AND METHODS METHODS
A retrospective study (2009-2019) was conducted on 92 patients with primary ovarian, fallopian tube or peritoneal HGEOC who underwent NACT followed by cytoreductive surgery. Nestin expression in tissue samples was semi-quantitatively evaluated defining nestin positivity for those with histochemical score ≥30. We then evaluated the prognostic value of nestin expression.
RESULTS RESULTS
The median progression-free survival was similar between nestin-positive (22 months) and nestin-negative (19 months) groups (p=0.57). Interestingly, the median overall survival was shorter for the nestin-positive group (48 vs. 67 months, respectively), however the difference did not reach statistical significance (p=0.43).
CONCLUSION CONCLUSIONS
Tissue nestin expression does not appear to be a relevant prognostic biomarker in HGEOC treated by NACT. However, we believe that prospective studies in larger cohorts should be conducted and evaluation of nestin in pre-NACT HGEOC samples needs to be explored.

Identifiants

pubmed: 35790290
pii: 42/7/3583
doi: 10.21873/anticanres.15845
doi:

Substances chimiques

Biomarkers 0
Nestin 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

3583-3594

Informations de copyright

Copyright © 2022 International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Auteurs

Hugo Veyssière (H)

University of Clermont Auvergne, INSERM, U1240, Molecular Imaging and Theranostic Strategies, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France; Hugo.VEYSSIERE@clermont.unicancer.fr.
Clinical Research Division, Clinical Research and Innovation Delegation, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France.
Clinical Investigation Center, UMR501, Clermont-Ferrand, France.

Ghassan Aldarazi (G)

Clinical Research Division, Clinical Research and Innovation Delegation, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France.
Clinical Investigation Center, UMR501, Clermont-Ferrand, France.
Department of Medical Oncology, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France.

Ioana Molnar (I)

University of Clermont Auvergne, INSERM, U1240, Molecular Imaging and Theranostic Strategies, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France.
Clinical Research Division, Clinical Research and Innovation Delegation, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France.
Clinical Investigation Center, UMR501, Clermont-Ferrand, France.

Xavier Durando (X)

University of Clermont Auvergne, INSERM, U1240, Molecular Imaging and Theranostic Strategies, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France.
Clinical Research Division, Clinical Research and Innovation Delegation, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France.
Clinical Investigation Center, UMR501, Clermont-Ferrand, France.
Department of Medical Oncology, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France.

Nina Radosevic-Robin (N)

Department of Anatomy and Pathological Cytology, Jean Perrin Comprehensive Cancer Center, Clermont-Ferrand, France.

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