An integrated atlas of human placental development delineates essential regulators of trophoblast stem cells.


Journal

Development (Cambridge, England)
ISSN: 1477-9129
Titre abrégé: Development
Pays: England
ID NLM: 8701744

Informations de publication

Date de publication:
01 07 2022
Historique:
received: 09 09 2021
accepted: 12 05 2022
entrez: 6 7 2022
pubmed: 7 7 2022
medline: 9 7 2022
Statut: ppublish

Résumé

The trophoblast lineage safeguards fetal development by mediating embryo implantation, immune tolerance, nutritional supply and gas exchange. Human trophoblast stem cells (hTSCs) provide a platform to study lineage specification of placental tissues; however, the regulatory network controlling self-renewal remains elusive. Here, we present a single-cell atlas of human trophoblast development from zygote to mid-gestation together with single-cell profiling of hTSCs. We determine the transcriptional networks of trophoblast lineages in vivo and leverage probabilistic modelling to identify a role for MAPK signalling in trophoblast differentiation. Placenta- and blastoid-derived hTSCs consistently map between late trophectoderm and early cytotrophoblast, in contrast to blastoid-trophoblast, which correspond to trophectoderm. We functionally assess the requirement of the predicted cytotrophoblast network in an siRNA-screen and reveal 15 essential regulators for hTSC self-renewal, including MAZ, NFE2L3, TFAP2C, NR2F2 and CTNNB1. Our human trophoblast atlas provides a powerful analytical resource to delineate trophoblast cell fate acquisition, to elucidate transcription factors required for hTSC self-renewal and to gauge the developmental stage of in vitro cultured cells.

Identifiants

pubmed: 35792865
pii: 275917
doi: 10.1242/dev.200171
pmc: PMC9340556
pii:
doi:

Substances chimiques

Basic-Leucine Zipper Transcription Factors 0
NFE2L3 protein, human 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 206684/Z/17/Z
Pays : United Kingdom
Organisme : Wellcome Trust
ID : WT RG89228
Pays : United Kingdom

Informations de copyright

© 2022. Published by The Company of Biologists Ltd.

Déclaration de conflit d'intérêts

Competing interests The authors declare no competing or financial interests.

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Auteurs

Yutong Chen (Y)

Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Site, Cambridge CB2 3EG, UK.
Centre for Trophoblast Research, University of Cambridge, Downing Site, Cambridge CB2 3EG, UK.
Wellcome Trust - Medical Research Council Stem Cell Institute, University of Cambridge, Jeffrey Cheah Biomedical Centre, Puddicombe Way, Cambridge CB2 0AW, UK.

Dylan Siriwardena (D)

Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Site, Cambridge CB2 3EG, UK.
Centre for Trophoblast Research, University of Cambridge, Downing Site, Cambridge CB2 3EG, UK.
Wellcome Trust - Medical Research Council Stem Cell Institute, University of Cambridge, Jeffrey Cheah Biomedical Centre, Puddicombe Way, Cambridge CB2 0AW, UK.

Christopher Penfold (C)

Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Site, Cambridge CB2 3EG, UK.
Centre for Trophoblast Research, University of Cambridge, Downing Site, Cambridge CB2 3EG, UK.
Wellcome Trust - Medical Research Council Stem Cell Institute, University of Cambridge, Jeffrey Cheah Biomedical Centre, Puddicombe Way, Cambridge CB2 0AW, UK.

Adam Pavlinek (A)

King's College London, Strand, London WC2R 2LS, UK.

Thorsten E Boroviak (TE)

Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Site, Cambridge CB2 3EG, UK.
Centre for Trophoblast Research, University of Cambridge, Downing Site, Cambridge CB2 3EG, UK.
Wellcome Trust - Medical Research Council Stem Cell Institute, University of Cambridge, Jeffrey Cheah Biomedical Centre, Puddicombe Way, Cambridge CB2 0AW, UK.

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