Innate Immune Responses by Respiratory Viruses, Including Rhinovirus, During Asthma Exacerbation.
bronchial asthma
eosinophils
epithelial cell-related cytokines
innate lymphoid cells
rhinovirus
Journal
Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960
Informations de publication
Date de publication:
2022
2022
Historique:
received:
30
01
2022
accepted:
13
05
2022
entrez:
7
7
2022
pubmed:
8
7
2022
medline:
9
7
2022
Statut:
epublish
Résumé
Viral infection, especially with rhinovirus (RV), is a major cause of asthma exacerbation. The production of anti-viral cytokines such as interferon (IFN)-β and IFN-α from epithelial cells or dendritic cells is lower in patients with asthma or those with high IgE, which can contribute to viral-induced exacerbated disease in these patients. As for virus-related factors, RV species C (RV-C) induces more exacerbated disease than other RVs, including RV-B. Neutrophils activated by viral infection can induce eosinophilic airway inflammation through different mechanisms. Furthermore, virus-induced or virus-related proteins can directly activate eosinophils. For example, CXCL10, which is upregulated during viral infection, activates eosinophils
Identifiants
pubmed: 35795686
doi: 10.3389/fimmu.2022.865973
pmc: PMC9250977
doi:
Substances chimiques
Antiviral Agents
0
Cytokines
0
Interferon-alpha
0
Interleukin-33
0
Interferon-beta
77238-31-4
Types de publication
Journal Article
Review
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
865973Informations de copyright
Copyright © 2022 Nakagome and Nagata.
Déclaration de conflit d'intérêts
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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