Pressure Overload Activates DNA-Damage Response in Cardiac Stromal Cells: A Novel Mechanism Behind Heart Failure With Preserved Ejection Fraction?

DNA damage response (DDR) cardiac mesenchymal stromal cells heart failure heart failure with preserved ejection fraction (HFpEF) mechanical stretch effects pressure overload

Journal

Frontiers in cardiovascular medicine
ISSN: 2297-055X
Titre abrégé: Front Cardiovasc Med
Pays: Switzerland
ID NLM: 101653388

Informations de publication

Date de publication:
2022
Historique:
received: 17 02 2022
accepted: 17 05 2022
entrez: 11 7 2022
pubmed: 12 7 2022
medline: 12 7 2022
Statut: epublish

Résumé

Heart failure with preserved ejection fraction (HFpEF) is a heterogeneous syndrome characterized by impaired left ventricular (LV) diastolic function, with normal LV ejection fraction. Aortic valve stenosis can cause an HFpEF-like syndrome by inducing sustained pressure overload (PO) and cardiac remodeling, as cardiomyocyte (CM) hypertrophy and fibrotic matrix deposition. Recently,

Identifiants

pubmed: 35811699
doi: 10.3389/fcvm.2022.878268
pmc: PMC9259931
doi:

Types de publication

Journal Article

Langues

eng

Pagination

878268

Informations de copyright

Copyright © 2022 Stadiotti, Santoro, Scopece, Pirola, Guarino, Polvani, Maione, Ascione, Li, Delia, Foiani, Pompilio and Sommariva.

Déclaration de conflit d'intérêts

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Ilaria Stadiotti (I)

Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.

Rosaria Santoro (R)

Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.
Department of Electronics, Information and Biomedical Engineering, Politecnico di Milano, Milan, Italy.

Alessandro Scopece (A)

Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.

Sergio Pirola (S)

Department of Cardiovascular Surgery, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.

Anna Guarino (A)

Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.

Gianluca Polvani (G)

Department of Cardiovascular Surgery, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.
Cardiovascular Tissue Bank of Milan, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.
Department of Biomedical, Surgical and Dental Sciences, Università degli Studi di Milano, Milan, Italy.

Angela Serena Maione (AS)

Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.

Flora Ascione (F)

IFOM (Istituto FIRC di Oncologia Molecolare), Milan, Italy.

Qingsen Li (Q)

IFOM (Istituto FIRC di Oncologia Molecolare), Milan, Italy.

Domenico Delia (D)

IFOM (Istituto FIRC di Oncologia Molecolare), Milan, Italy.

Marco Foiani (M)

IFOM (Istituto FIRC di Oncologia Molecolare), Milan, Italy.
Department of Oncology and Hematology-Oncology, Università degli Studi di Milano, Milan, Italy.

Giulio Pompilio (G)

Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.
Department of Biomedical, Surgical and Dental Sciences, Università degli Studi di Milano, Milan, Italy.

Elena Sommariva (E)

Vascular Biology and Regenerative Medicine Unit, Centro Cardiologico Monzino IRCCS (Istituto di Ricovero e Cura a Carattere Scientifico), Milan, Italy.

Classifications MeSH