Hacking Commensal Bacteria to Consolidate the Adaptive Mucosal Immune Response in the Gut-Lung Axis: Future Possibilities for SARS-CoV-2 Protection.
COVID-19
epitope
gut microbiome
mucosal immunology
mucosal pathogens
mucosal vaccines
Journal
Biotech (Basel (Switzerland))
ISSN: 2673-6284
Titre abrégé: BioTech (Basel)
Pays: Switzerland
ID NLM: 9918383086206676
Informations de publication
Date de publication:
30 Jan 2022
30 Jan 2022
Historique:
received:
05
11
2021
revised:
04
01
2022
accepted:
26
01
2022
entrez:
13
7
2022
pubmed:
14
7
2022
medline:
14
7
2022
Statut:
epublish
Résumé
Infectious diseases caused by mucosal pathogens significantly increase mortality and morbidity. Thus, the possibility to target these pathogens at their primary entry points can consolidate protective immunity. Regarding SARS-CoV-2 infection, it has been observed that the upper respiratory mucosa is highly affected and that dysregulation of resident microbiota in the gut-lung axis plays a crucial role in determining symptom severity. Thus, understanding the possibility of eliciting various mucosal and adaptive immune responses allows us to effectively design bacterial mucosal vaccine vectors. Such design requires rationally selecting resident bacterial candidates as potential host carriers, evaluating effective carrier proteins for stimulating an immune response, and combining these two to improve antigenic display and immunogenicity. This review investigated mucosal vaccine vectors from 2015 to present, where a few have started to utilize
Identifiants
pubmed: 35822811
pii: biotech11010003
doi: 10.3390/biotech11010003
pmc: PMC9245903
pii:
doi:
Types de publication
Journal Article
Review
Langues
eng
Subventions
Organisme : Swedish Research Council/Vetenskapsrådet
ID : 2020-06320
Organisme : National Research Foundation of Korea
ID : 2020K1A3A1A47110820
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