Lurbinectedin in patients with pretreated neuroendocrine tumours: Results from a phase II basket study.


Journal

European journal of cancer (Oxford, England : 1990)
ISSN: 1879-0852
Titre abrégé: Eur J Cancer
Pays: England
ID NLM: 9005373

Informations de publication

Date de publication:
Sep 2022
Historique:
received: 24 01 2022
revised: 03 06 2022
accepted: 10 06 2022
pubmed: 14 7 2022
medline: 17 8 2022
entrez: 13 7 2022
Statut: ppublish

Résumé

Patients with neuroendocrine tumours (NETs) need alternative therapies after failure of first-line therapy. This phase II trial evaluated lurbinectedin, a selective inhibitor of oncogenic transcription, at 3.2 mg/m Two of 31 evaluable patients had confirmed partial responses (ORR = 6.5%; 95%CI, 0.8-21.4%). Median DoR was 4.7 months (95% CI, 4.0-5.4 months), median PFS was 1.4 months (95% CI, 1.2-3.0 months) and median OS was 7.4 months (95% CI, 3.4-16.2 months). Lurbinectedin showed an acceptable, predictable and manageable safety profile. The most common grade 3/4 toxicity was neutropenia (40.6%; grade 4, 12.4%; febrile neutropenia, 3.1%). Considering the exploratory aim of this trial that evaluated a heterogeneous population of NETs patients, and the signs of antitumour activity observed (two confirmed partial responses and seven long disease stabilisations), further development of lurbinectedin is warranted in a more selected NETs population. Sponsor Study Code: PM1183-B-005-14. EudraCT number: 2014-003773-42. gov reference: NCT02454972.

Sections du résumé

BACKGROUND
Patients with neuroendocrine tumours (NETs) need alternative therapies after failure of first-line therapy.
PATIENTS AND METHODS
This phase II trial evaluated lurbinectedin, a selective inhibitor of oncogenic transcription, at 3.2 mg/m
RESULTS
Two of 31 evaluable patients had confirmed partial responses (ORR = 6.5%; 95%CI, 0.8-21.4%). Median DoR was 4.7 months (95% CI, 4.0-5.4 months), median PFS was 1.4 months (95% CI, 1.2-3.0 months) and median OS was 7.4 months (95% CI, 3.4-16.2 months). Lurbinectedin showed an acceptable, predictable and manageable safety profile. The most common grade 3/4 toxicity was neutropenia (40.6%; grade 4, 12.4%; febrile neutropenia, 3.1%).
CONCLUSIONS
Considering the exploratory aim of this trial that evaluated a heterogeneous population of NETs patients, and the signs of antitumour activity observed (two confirmed partial responses and seven long disease stabilisations), further development of lurbinectedin is warranted in a more selected NETs population.
TRIAL REGISTRATION NUMBER
Sponsor Study Code: PM1183-B-005-14. EudraCT number: 2014-003773-42.
CLINICALTRIALS
gov reference: NCT02454972.

Identifiants

pubmed: 35830841
pii: S0959-8049(22)00375-6
doi: 10.1016/j.ejca.2022.06.024
pii:
doi:

Substances chimiques

Carbolines 0
Heterocyclic Compounds, 4 or More Rings 0
PM 01183 0

Banques de données

ClinicalTrials.gov
['NCT02454972']

Types de publication

Clinical Trial, Phase II Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

340-348

Informations de copyright

Copyright © 2022 The Author(s). Published by Elsevier Ltd.. All rights reserved.

Déclaration de conflit d'intérêts

Conflict of interest statement The authors declare the following financial interests/personal relationships which may be considered as potential competing interests: Cristian Fernández, Carmen Kahatt, Vicente Alfaro, Mariano Siguero and Ali Zeaiter are permanent employees of PharmaMar. Victor Moreno reports consulting fees from Roche, Bayer, BMS, Janssen and Basilea. Vivek Subbiah reports grants from Pharmamar, Eli Lilly/LOXO Oncology, Blueprint Medicines Corporation, Turning Point Therapeutics, Boston Pharmaceuticals; and grants from Helsinn Pharmaceuticals during the conduct of the study; in addition, Vivek Subbiah reports a grant and advisory board/consultant position with Eli Lilly/Loxo Oncology during the conduct of the study; research grants from Roche/Genentech, Bayer, GlaxoSmithKline, Nanocarrier, Vegenics, Celgene, Northwest Biotherapeutics, Berghealth, Incyte, Fujifilm, D3, Pfizer, Multivir, Amgen, Abbvie, Alfa-sigma, Agensys, Boston Biomedical, Idera Pharma, Inhibrx, Exelixis, Blueprint Medicines, Altum, Dragonfly Therapeutics, Takeda, National Comprehensive Cancer Network, NCI-CTEP, University of Texas MD Anderson Cancer Center, Turning Point Therapeutics, Boston Pharmaceuticals, Novartis, Pharmamar, Medimmune; an advisory board/consultant position with Helsinn, Incyte, QED Pharma, Daiichi-Sankyo, Signant Health, Novartis, Janssen, Relay Therapeutics, Roche, Medimmune; travel funds from Pharmamar, Incyte, ASCO, ESMO; other support from Medscape; all outside the submitted work. No disclosures were reported by the other authors.

Auteurs

Federico Longo-Muñoz (F)

Medical Oncology, Hospital Ramón y Cajal, Madrid, Spain.

Daniel Castellano (D)

Medical Oncology, Hospital Universitario 12 de Octubre, Madrid, Spain.

Jerome Alexandre (J)

Medical Oncology, Cochin Hospital, AP-HP, Paris, France.

Sant P Chawla (SP)

Medical Oncology, Sarcoma Oncology Center, Santa Monica CA 90403, USA.

Cristian Fernández (C)

Clinical R&D, PharmaMar, Colmenar Viejo, Spain.

Carmen Kahatt (C)

Clinical R&D, PharmaMar, Colmenar Viejo, Spain.

Vicente Alfaro (V)

Clinical R&D, PharmaMar, Colmenar Viejo, Spain.

Mariano Siguero (M)

Clinical R&D, PharmaMar, Colmenar Viejo, Spain.

Ali Zeaiter (A)

Clinical R&D, PharmaMar, Colmenar Viejo, Spain.

Victor Moreno (V)

Medical Oncology, START Madrid-FJD, Hospital Fundación Jiménez Díaz, Madrid, Spain.

Enrique Sanz-García (E)

Medical Oncology, Vall d'Hebron Hospital Campus and Institute of Oncology (VHIO), Barcelona, Spain.

Ahmad Awada (A)

Medical Oncology, Institut Jules Bordet, Université Libre De Bruxelles, Brussels, Belgium.

Ana Santaballa (A)

Medical Oncology, Hospital La Fe, Valencia, Spain.

Vivek Subbiah (V)

Medical Oncology, UT MD Anderson Cancer Center, Houston, USA. Electronic address: vsubbiah@mdanderson.org.

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Classifications MeSH