Adeno-Associated Virus-Mediated Gene Therapy for Patients' Fibroblasts, Induced Pluripotent Stem Cells, and a Mouse Model of Congenital Adrenal Hyperplasia.


Journal

Human gene therapy
ISSN: 1557-7422
Titre abrégé: Hum Gene Ther
Pays: United States
ID NLM: 9008950

Informations de publication

Date de publication:
08 2022
Historique:
pubmed: 16 7 2022
medline: 19 8 2022
entrez: 15 7 2022
Statut: ppublish

Résumé

Congenital adrenal hyperplasia (CAH) is an autosomal recessive disorder caused by steroidogenic enzymes containing monogenetic defects. Most steroidogenic enzymes are cytochrome P450 groups that can be categorized as microsomal P450s, including 21-hydroxylase and 17α-hydroxylase/17,20 lyase, and mitochondrial P450s, including 11β-hydroxylase. It has been shown that ectopic administration of

Identifiants

pubmed: 35838129
doi: 10.1089/hum.2022.005
doi:

Substances chimiques

Steroid 21-Hydroxylase EC 1.14.14.16
Steroid 17-alpha-Hydroxylase EC 1.14.14.19
Steroid 11-beta-Hydroxylase EC 1.14.15.4

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

801-809

Auteurs

Yasuhiro Naiki (Y)

Division of Endocrinology and Metabolism, National Center for Child Health and Development, Tokyo, Japan.

Mami Miyado (M)

Department of Molecular Endocrinology, National Research Institute for Child Health and Development, Tokyo, Japan.

Miyuki Shindo (M)

Division of Laboratory Animal Resources, National Research Institute for Child Health and Development, Tokyo, Japan; and.

Reiko Horikawa (R)

Division of Endocrinology and Metabolism, National Center for Child Health and Development, Tokyo, Japan.

Yuichi Hasegawa (Y)

Division of Urology, National Center for Child Health and Development, Tokyo, Japan.

Noriyuki Katsumata (N)

Department of Molecular Endocrinology, National Research Institute for Child Health and Development, Tokyo, Japan.

Shuji Takada (S)

Department of Systems BioMedicine, National Research Institute for Child Health and Development, Tokyo, Japan.

Hidenori Akutsu (H)

Department of Reproductive Medicine, National Research Institute for Child Health and Development, Tokyo, Japan.

Masafumi Onodera (M)

Department of Human Genetics, National Research Institute for Child Health and Development, Tokyo, Japan.

Maki Fukami (M)

Department of Molecular Endocrinology, National Research Institute for Child Health and Development, Tokyo, Japan.

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Classifications MeSH