Circulating cardiac biomarkers improve risk stratification for incident cardiovascular disease in community dwelling populations.


Journal

EBioMedicine
ISSN: 2352-3964
Titre abrégé: EBioMedicine
Pays: Netherlands
ID NLM: 101647039

Informations de publication

Date de publication:
Aug 2022
Historique:
received: 23 02 2022
revised: 28 06 2022
accepted: 01 07 2022
pubmed: 20 7 2022
medline: 17 8 2022
entrez: 19 7 2022
Statut: ppublish

Résumé

Plasma cardiac markers may assist in prediction of incident cardiovascular disease. The incremental value of cardiac Troponins (T and I) and NT-proBNP added to risk factors in the PREDICT score for incident cardiovascular disease (CVD) in primary care, was assessed in 4102 asymptomatic participants in a randomised controlled trial of Vitamin D (ViDA). Findings were corroborated in 2528 participants in a separate community-based observational registry of CVD-free volunteers (HVOLS). Hazard ratios for first cardiovascular events adjusted for PREDICT risk factors, comparing fifth to first quintiles of marker plasma concentrations, were 2.57 (95% CI 1.47-4.49); 3.01 (1.66-5.48) and 3.38 (2.04-5.60) for hs-cTnI, hs-cTnT and NT-proBNP respectively. The C statistic for discrimination of the primary endpoint increased from 0.755 to 0.771 (+0.016, p = 0.01). Cardiac marker data correctly reclassified risk upwards in 6.7% of patients and downwards in 3.3%. These findings were corroborated by results from HVOLS. Increments in plasma cardiac biomarkers robustly and reproducibly predicted increased hazard of incident CVD, independent of established risk factors, in two community-dwelling populations. Cardiac markers may augment risk assessment for onset of CVD in primary care. ViDA was funded by the Health Research Council of New Zealand (grant 10/400) and the Accident Compensation Corporation. HVOLS was funded by the Health Research Council of NZ Programme Grants (grants 02/152 and 08/070) and by grants from the Heart Foundation of NZ and the Christchurch Heart Institute Trust. Roche Diagnostics provided in-kind support for NT-proBNP and hs-cTnT assays and Abbott Laboratories for hs-cTnI assays.

Sections du résumé

BACKGROUND BACKGROUND
Plasma cardiac markers may assist in prediction of incident cardiovascular disease.
METHODS METHODS
The incremental value of cardiac Troponins (T and I) and NT-proBNP added to risk factors in the PREDICT score for incident cardiovascular disease (CVD) in primary care, was assessed in 4102 asymptomatic participants in a randomised controlled trial of Vitamin D (ViDA). Findings were corroborated in 2528 participants in a separate community-based observational registry of CVD-free volunteers (HVOLS).
FINDINGS RESULTS
Hazard ratios for first cardiovascular events adjusted for PREDICT risk factors, comparing fifth to first quintiles of marker plasma concentrations, were 2.57 (95% CI 1.47-4.49); 3.01 (1.66-5.48) and 3.38 (2.04-5.60) for hs-cTnI, hs-cTnT and NT-proBNP respectively. The C statistic for discrimination of the primary endpoint increased from 0.755 to 0.771 (+0.016, p = 0.01). Cardiac marker data correctly reclassified risk upwards in 6.7% of patients and downwards in 3.3%. These findings were corroborated by results from HVOLS.
INTERPRETATION CONCLUSIONS
Increments in plasma cardiac biomarkers robustly and reproducibly predicted increased hazard of incident CVD, independent of established risk factors, in two community-dwelling populations. Cardiac markers may augment risk assessment for onset of CVD in primary care.
FUNDING BACKGROUND
ViDA was funded by the Health Research Council of New Zealand (grant 10/400) and the Accident Compensation Corporation. HVOLS was funded by the Health Research Council of NZ Programme Grants (grants 02/152 and 08/070) and by grants from the Heart Foundation of NZ and the Christchurch Heart Institute Trust. Roche Diagnostics provided in-kind support for NT-proBNP and hs-cTnT assays and Abbott Laboratories for hs-cTnI assays.

Identifiants

pubmed: 35850010
pii: S2352-3964(22)00351-6
doi: 10.1016/j.ebiom.2022.104170
pmc: PMC9294489
pii:
doi:

Substances chimiques

Biomarkers 0
Peptide Fragments 0
Troponin I 0
Troponin T 0
Natriuretic Peptide, Brain 114471-18-0
Vitamin D 1406-16-2

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

104170

Informations de copyright

Copyright © 2022 The Authors. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of interests Zhenqiang Wu: ViDA was funded by the Health Research Council of New Zealand (grant 10/400) and the Accident Compensation Corporation. HVOLs was funded by the Health Research Council of NZ Programme Grants (grants 02/152 and 08/070) and by grants from the Heart Foundation of NZ and the Christchurch Heart Institute Trust. Roche Diagnostics provided in-kind support for NT-proBNP and hsTnT assays and Abbott Laboratories for hsTnI assays. Anna P Pilbrow: Nil to disclose. Oi Wah Liew: Nil to disclose. Jenny P C Chong: Nil to disclose. John Sluyter: Support from Christchurch Heart Institute; travel costs for traveling to Christchurch to discuss ViDA study (give presentation) and methodology; costs for working with plasma aliquots from ViDA study. Data preparation, extraction of samples from freezer, data checking, arranging samples for couriering. Lynley K Lewis: Nil to disclose. Moritz Lasse: Nil to disclose. Chris M Frampton: Nil to disclose. Rod Jackson: Nil to disclose. Katrina Poppe: New Zealand Heart Foundation Senior fellowship to support salary; domestic travel to national meetings. Carlos Arturo Camargo Jr: Nil to disclose. Vicky Cameron A.: Nil to disclose. Robert Scragg: Health Research Council of New Zealand Application 10/400. Main government funder of health research in NZ. Accident Compensation Corporation. Main government insurer for health costs arising from injury. A Mark Richards: Roche Diagnostics In kind support for assays. Abbott Labs In kind support for assays. Roche Diagnostics advisory board fees.

Auteurs

Zhenqiang Wu (Z)

School of Population Health, University of Auckland, Auckland, New Zealand.

Anna P Pilbrow (AP)

Christchurch Heart Institute, University of Otago, New Zealand.

Oi Wah Liew (OW)

Cardiovascular Research Institute, National University of Singapore, Singapore.

Jenny P C Chong (JPC)

Cardiovascular Research Institute, National University of Singapore, Singapore.

John Sluyter (J)

School of Population Health, University of Auckland, Auckland, New Zealand.

Lynley K Lewis (LK)

Christchurch Heart Institute, University of Otago, New Zealand.

Moritz Lasse (M)

Christchurch Heart Institute, University of Otago, New Zealand.

Chris M Frampton (CM)

Christchurch Heart Institute, University of Otago, New Zealand.

Rod Jackson (R)

School of Population Health, University of Auckland, Auckland, New Zealand.

Katrina Poppe (K)

School of Population Health, University of Auckland, Auckland, New Zealand.

Carlos Arturo Camargo (CA)

Department of Emergency Medicine, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.

Vicky A Cameron (VA)

Christchurch Heart Institute, University of Otago, New Zealand.

Robert Scragg (R)

School of Population Health, University of Auckland, Auckland, New Zealand.

A Mark Richards (AM)

Christchurch Heart Institute, University of Otago, New Zealand; Cardiovascular Research Institute, National University of Singapore, Singapore. Electronic address: mark.richards@cdhb.health.nz.

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Classifications MeSH